E. A. Gault
University of Glasgow
27 Papers
103 Citations
E. A. Gault is an academic researcher from University of Glasgow. The author has contributed to research in topics: Bovine papillomavirus & Telomere. The author has an hindex of 13, co-authored 27 publications.
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Papers
Real-time quantitative PCR assay for measurement of avian telomeres
François Criscuolo,Pierre Bize,Lubna Nasir,Neil B. Metcalfe,Christopher Foote,Kate Griffiths,E. A. Gault,Pat Monaghan +7 more
TL;DR: This PCR assay for measurement of bird telomeres, which is fast and requires only small amounts of genomic DNA, should open new avenues in the study of environmental factors influencing variation in telomere length, and how this variation translates into variation in cellular and whole organism senescence.
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Bovine papillomavirus infection in equine sarcoids and in bovine bladder cancers.
TL;DR: Rolling circle amplification demonstrated that BPV-1 andBPV-2 genomes exist as double stranded, episomal, circular forms in the two tumours, and realtime quantitative PCR revealed that equine sarcoids contained higher viral DNA loads compared to bovine bladder cancers.
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Equine telomeres and telomerase in cellular immortalisation and ageing.
TL;DR: It would appear that telomerase does not play a major role in the development of the most common benign tumours of the horse, and telomere attrition may contribute to ageing in equids.
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Establishment and characterization of equine fibroblast cell lines transformed in vivo and in vitro by BPV-1: model systems for equine sarcoids
TL;DR: These findings confirm that the virus has an active role in the induction of sarcoids and the lines will be invaluable for further studies on the role of BPV-1 in sarcoid pathology.
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Telomere dynamics in relation to early growth conditions in the wild in the lesser black-backed gull
TL;DR: Examination of telomere dynamics during growth under natural conditions in the lesser black-backed gull Larus fuscus showed a strong relationship between telomeres length at hatching and at 10 days old, demonstrating that the variation in hatching telomer length caused by embryonic growth conditions remained consistent during the initial post-hatching period.
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