Domenico Ribatti
University of Bari
965 Papers
6.1K Citations
Domenico Ribatti is an academic researcher from University of Bari. The author has contributed to research in topics: Angiogenesis & Biology. The author has an hindex of 96, co-authored 913 publications. Previous affiliations of Domenico Ribatti include University of Genoa & National Institutes of Health.
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Papers
Involvement of caspase 8 and c-FLIPL in the proangiogenic effects of the tumour necrosis factor-related apoptosis-inducing ligand (TRAIL).
Giuseppina Cantarella,Giulia Di Benedetto,Domenico Ribatti,Gloria Saccani-Jotti,Renato Bernardini +4 more
TL;DR: The data suggest that the non‐apoptotic effects of TRAIL include mitogenesis and increased mobility of endothelial cells, and eventually angiogenesis, suggesting that TRAIL may represent a target for pharmacological manipulation.
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Inflammatory Infiltrate and Angiogenesis in Mantle Cell Lymphoma.
Tiziana Annese,Giuseppe Ingravallo,Roberto Tamma,Michelina De Giorgis,Eugenio Maiorano,Tommasina Perrone,Francesco Albano,Giorgina Specchia,Domenico Ribatti +8 more
TL;DR: The data indicate that, in MCL, SOX11 expression is associated with increased angiogenesis and a high CD4+ and CD8+ T-cell infiltration, which are not sustained by CD163+ macrophages infiltrate and p53 expression.
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Stat3-positive tumor cells contribute to vessels neoformation in primary central nervous system lymphoma
Simona Ruggieri,Roberto Tamma,Nicoletta Resta,Francesco Albano,Nicoletta Coccaro,Daria Carmela Loconte,Tiziana Annese,Mariella Errede,Giorgina Specchia,Rebecca Senetta,Paola Cassoni,Domenico Ribatti,Beatrice Nico +12 more
TL;DR: Investigating Stat3 mutations in lymphoma cells to clarify the role of the constitutive expression of Stat3 and of its phosphorylated forms showed that in PCNSL, putative endothelial cells lining the vessels are heterogeneous, expressing FVIII/ pStat3/CD133 (presumably originally they are vascular progenitor cells).
The adaptor protein p66Shc is a positive regulator in the angiogenic response induced by hypoxic T cells.
Antonella Naldini,Emilia Morena,Annalisa Pucci,Michela Pellegrini,Cosima T. Baldari,Pier Giuseppe Pelicci,Marco Presta,Domenico Ribatti,Fabio Carraro +8 more
TL;DR: It is reported that VEGF represents the major proangiogenic factor expressed by T cells exposed to hypoxia, a common feature of inflammation and tumor microenvironment, and hypoxic T cells may contribute to the onset of angiogenesis through a novel V EGF‐mediated mechanism.
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