Domenico Ribatti
University of Bari
965 Papers
6.1K Citations
Domenico Ribatti is an academic researcher from University of Bari. The author has contributed to research in topics: Angiogenesis & Biology. The author has an hindex of 96, co-authored 913 publications. Previous affiliations of Domenico Ribatti include University of Genoa & National Institutes of Health.
Chat about Author
Papers
Crosstalk between angiogenesis and lymphangiogenesis in tumor progression
TL;DR: The literature concerning the crosstalk between angiogenesis and lymphangiogenesis in tumor progression is summarized, that is, involvement of VEGF-C, V EGF-D and VEGFR-3 inAngiogenesis, and the role played by VEGf-A and Ang-2 in lymphang iogenesis, respectively.
Max D. Cooper and the Delineation of Two Lymphoid Lineages in the Adaptive Immune System
TL;DR: This article outlines the fundamental contribution of Max D. Cooper to the analysis of the role of the thymus and of the bursa of Fabricius in the development of immunologic competence both before and after birth.
The development of human mast cells. An historical reappraisal
TL;DR: The understanding of mast cell (MC) differentiation is derived mainly from in vitro studies of different stages of stem and progenitor cells.
Progression of mycosis fungoides is associated with changes in angiogenesis and expression of the matrix metalloproteinases 2 and 9
Angelo Vacca,Silvia Moretti,Domenico Ribatti,Antonio Pellegrino,Nicola Pimpinelli,Beatrice Bianchi,E. Bonifazi,Roberto Ria,Gabriella Serio,F. Dammacco +9 more
TL;DR: In this paper, 57 biopsies of mycosis fungoides (MF), a haematological tumour of T-cell lineage, were investigated immunohistochemically for the extent of angiogenesis, and by in situ hybridisation for the expression of matrix metalloproteinases 2 (MMP-2, collagenase A) and 9 (mMP-9), collagenase B).
Osteopontin (Eta-1) and Fibroblast Growth Factor-2 Cross-Talk in Angiogenesis
Daria Leali,Patrizia Dell'Era,Helena Stabile,Barbara Sennino,Ann F. Chambers,Antonella Naldini,Silvano Sozzani,Beatrice Nico,Domenico Ribatti,Marco Presta +9 more
TL;DR: OPN-mediated recruitment of proangiogenic monocytes may represent a mechanism of amplification of FGF2-induced neovascularization during inflammation, wound healing, and tumor growth.