Domenico Ribatti
University of Bari
965 Papers
6.1K Citations
Domenico Ribatti is an academic researcher from University of Bari. The author has contributed to research in topics: Angiogenesis & Biology. The author has an hindex of 96, co-authored 913 publications. Previous affiliations of Domenico Ribatti include University of Genoa & National Institutes of Health.
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Papers
Pyogenic Granuloma Stimulates Angiogenesis in the Chick Embryo Chorioallantoic Membrane
Domenico Ribatti,Angelo Vacca,G. Schiraldi,S. Sorino,F. Caprio,Francesco Mazzotta,Luisa Roncali,E. Bonifazi +7 more
TL;DR: The role played in angiogenic response by the inflammatory cells, mainly mast cells, forming the perilesional infiltrate was supported by this study.
10
Aristotle: the first student of angiogenesis
TL;DR: The cardiovascular system is the first functional organ system to develop in the vertebrate embryo and several observations indicate that vasculogenesis may not be restricted to early embryogenesis, but may also have a physiological role or contribute to the pathology of vascular diseases in adults.
10
Serum insulin-like growth factor is not elevated in patients with early B-cell chronic lymphocytic leukemia but is still a prognostic factor for disease progression.
Stefano Molica,Gaetano Vitelli,Rosanna Mirabelli,Giovanna Digiesu,Diana Giannarelli,Antonio Cuneo,Domenico Ribatti,Angelo Vacca +7 more
TL;DR: Serum levels of IGF‐1 did not correlate with markers of tumor burden or clinical status in CLL thus suggesting that levels of this cytokine do not reflect the intrinsic malignancy of disease.
10
•Journal Article
Macrophages and mast cells are involved in carotid plaque instability.
TL;DR: Although advanced symptomatic and asymptomatic carotid plaques present similar histomorphological characteristics, the degree of macrophage and mast cell infiltration and differences in microvascular density could help to discriminate between symptomatic
10
Bone marrow vascular niche and the control of tumor growth in hematological malignancies.
TL;DR: Depletion of either osteoblasts or endothelial cells is negatively associated with HSC survival, suggesting that both niches are important for maintaining HSC.