Domenico Ribatti
University of Bari
965 Papers
6.1K Citations
Domenico Ribatti is an academic researcher from University of Bari. The author has contributed to research in topics: Angiogenesis & Biology. The author has an hindex of 96, co-authored 913 publications. Previous affiliations of Domenico Ribatti include University of Genoa & National Institutes of Health.
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Papers
Endogenous Basic Fibroblast Growth Factor Is Implicated in the Vascularization of the Chick Embryo Chorioallantoic Membrane
TL;DR: Evidence is provided indicating that endogenous bFGF has a rate-limiting role in the vascularization of the CAM during chick embryogenesis, rather than that present in the CAF, appears to be involved in this developmental process.
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The role of mast cells in tumour angiogenesis.
TL;DR: Human mast cells are conventionally divided into two types depending on the expression of different proteases in their granules, which are predominantly found in connective tissue areas, such as skin, conjunctiva and synovium.
160
•Journal Article
The role of podoplanin in tumor progression and metastasis
TL;DR: Podoplanin expression was found in tumor cells of various types of cancer, such as vascular tumors, malignant mesothelioma, tumors of the central nervous system (CNS), germ cell tumors and squamous cell carcinomas, and podoplanin significantly increases the detection of lymphovascular invasion in different types of malignant tumors.
156
Angiogenesis extent and macrophage density increase simultaneously with pathological progression in B-cell non-Hodgkin's lymphomas
Angelo Vacca,Domenico Ribatti,L Ruco,F Giacchetta,Beatrice Nico,Fabio Quondamatteo,Roberto Ria,Monica Iurlaro,F. Dammacco +8 more
TL;DR: Investigation of node biopsies of benign lymphadenopathies and B-cell non-Hodgkin’s lymphomas suggested that angiogenesis occurring in B-NHLs increases with tumour progression, and that macrophages promote the induction ofAngiogenesis via the release of their angiogenic factors.
In vivo activation of JAK2/STAT-3 pathway during angiogenesis induced by GM-CSF
TL;DR: These findings provide the first evidence that the JAK‐2/STAT‐3 pathway is activated in vivo and participates in vessel formation triggered by GM‐CSF.
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