Dirk C. G. Straver
Utrecht University
7 Papers
6 Citations
Dirk C. G. Straver is an academic researcher from Utrecht University. The author has contributed to research in topics: Multifocal motor neuropathy & Mismatch negativity. The author has an hindex of 5, co-authored 6 publications.
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Papers
Cold paresis in multifocal motor neuropathy
Dirk C. G. Straver,Jan-Thies H. van Asseldonk,Nicolette C. Notermans,John H. J. Wokke,Leonard H. van den Berg,Hessel Franssen +5 more
TL;DR: Symptoms of cold paresis are common in peripheral nervous system disorders, particularly in MMN, and this supports the hypothesis that cold plesis inMMN does not reflect demyelination only, but may indicate the existence of inflammatory nerve lesions with permanently depolarized axons that only just conduct at normal temperature, but fail at lower temperatures.
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Symptoms of activity-induced weakness in peripheral nervous system disorders.
TL;DR: Investigation by questionnaire in MMN and CIDP patients found activity‐induced weakness is frequently reported, however, not specific for these neuropathies as PSMA patients reported it to the same extent.
6
Plasticity of visual evoked potentials in patients with neurofibromatosis type 1
Jesminne Castricum,J. H. M. Tulen,A.M.F. Heuvelmans,Geert Geleijnse,Dirk C. G. Straver,Walter Taal,Steven A. Kushner,Ype Elgersma +7 more
TL;DR: In this article , the authors measured the increase of peak amplitudes C1, P1, and N1 induced by 10-min modulation of checkerboard reversals in 22 adult NF1 patients and 30 controls.
2
Spinal arteriovenous shunts presenting as intracranial subarachnoid haemorrhage
TL;DR: Investigation of the characteristics of patients with SAVS who present with intracranial SAH without symptoms and signs suggesting a spinal cause found one patient (0.05%, 95 % CI 0.006–0.3%).
Pathophysiology of immune-mediated demyelinating neuropathies--Part II: Neurology.
TL;DR: The relationship between pathophysiology and symptoms and thepathophysiology of specific disease entities are described and discussed, including Guillain–Barré syndrome, chronic inflammatory demyelinating polyneuropathy, multifocal motor neuropathy, anti–myelin‐associated glycoprotein neuropathies, and POEMS syndrome.