David Muir
University of California, San Diego
13 Papers
418 Citations
David Muir is an academic researcher from University of California, San Diego. The author has contributed to research in topics: Schwann cell & Nerve growth factor. The author has an hindex of 12, co-authored 13 publications.
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Papers
An enzyme-linked immunosorbent assay for bromodeoxyuridine incorporation using fixed microcultures
TL;DR: The ELISA measurement of BrdU incorporation compares favorably with measurements of tritiated thymidine incorporation and offers the additional advantages that the same microculture can be examined for cell number, for cell morphology, and for the percentage of cells having Brd U-labeled nuclei and other antigens.
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Laminin-like antigen in rat CNS neurons: Distribution and changes upon brain injury and nerve growth factor treatment
TL;DR: Medial septum neuronal immunoreactivity was lost after septo-hippocampal disconnection, but could be preserved or even restored by intraventricular administration of nerve growth factor and it remains to be determined whether CNS neuronal "laminin" processes activities similar to those found for laminin in vitro.
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The localization of nerve growth factor-like immunoreactivity in the adult rat basal forebrain and hippocampal formation.
TL;DR: The presence of an NGF‐like antigen in association with cholinergic neurons of the basal forebrain is demonstrated and strongly support the hypothesis that NGF may serve as an endogenous trophic factor for this adult neuronal population.
110
Schwannoma cell-derived inhibitor of the neurite-promoting activity of laminin.
TL;DR: The isolated Schwannoma-derived inhibitor suggests that the neurite-promoting activity of laminin is subject to regulation through association with a proteoglycan and entactin.
Autocrine inhibition of mitotic activity in cultured oligodendrocyte-type-2 astrocyte (O-2A) precursor cells
TL;DR: The results suggest that O‐2A cells secrete an autocrine inhibitory factor capable of reversibly withdrawing them from the cell cycle, which suggests that the commitment to the oligodendrocyte lineage might proceed through more than one discrete stage.
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