Daqiang He
Zhejiang University
6 Papers
28 Citations
Daqiang He is an academic researcher from Zhejiang University. The author has contributed to research in topics: Hippocampal formation & Apoptosis. The author has an hindex of 4, co-authored 4 publications.
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Papers
Endoplasmic reticulum stress-mediated hippocampal neuron apoptosis involved in diabetic cognitive impairment.
TL;DR: The present results suggested that the CHOP-dependent endoplasmic reticulum (ER) stress-mediated apoptosis may be involved in hyperglycemia-induced hippocampal synapses and neurons impairment and promote the diabetic cognitive impairment.
Effects of gestational isoflurane exposure on postnatal memory and learning in rats
TL;DR: It is demonstrated that gestational exposure to a clinically relevant concentration of isoflurane could cause neuron apoptosis, changes of synaptic structure, and postnatal spatial memory and learning impairments in offspring.
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Tanshinone IIA inhibits cardiomyocyte apoptosis and rescues cardiac function during doxorubicin-induced cardiotoxicity by activating the DAXX/MEK/ERK1/2 pathway.
TL;DR: In this article , Tan IIA (Tan IIA) was shown to significantly increase cell viability and protect against DOX-induced apoptosis, which was associated with extracellular signal-regulated protein kinase 1/2 (ERK1/2) and mitogenactivated protein kinases (MEK) expression.
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Protective effect of tanshinone IIA on rat kidneys during hypothermic preservation.
TL;DR: The results suggest that the supplementation of tanshinone IIA in standard Celsior solution may significantly improve long-term kidney preservation and that Attenuating oxidative stress injury and decreasing endoplasmic reticulum (ER) stress‑mediated apoptosis may play a role in the protection of kidney hypothermic preservation.
C/EBP homologous protein induces mesangial cell apoptosis under hyperglycemia.
TL;DR: Investigation of the role of C/EBP homologous protein (CHOP), an important protein in endoplasmic reticulum stress‑mediated mesangial cell apoptosis in hyperglycemia, indicates that CHOP mediates apoptotic cells under hyper glycemia and may play a role in the development of diabetic nephropathy.