Daphne Cornish
4 Papers
Daphne Cornish is an academic researcher. The author has contributed to research in topics: Biology & Medicine. The author has an hindex of 2, co-authored 4 publications.
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Papers
Circulating ACE2-expressing extracellular vesicles block broad strains of SARS-CoV-2
Lamiaa El-Shennawy,Andrew D. Hoffmann,Nurmaa Dashzeveg,Kathleen M. McAndrews,P.J. Mehl,Daphne Cornish,Zihao Yu,Valerie Tokars,Vlad Nicolaescu,Anastasia Tomatsidou,Chengsheng Mao,Christopher Felicelli,Chia-Feng Tsai,Carolina Ostiguin,Yuzhi Jia,Lin Li,Kevin Furlong,Jan Wysocki,Xin Luo,Carolina F. Ruivo,Daniel Batlle,Thomas J. Hope,Yang Shen,Young Kwang Chae,Hui Zhang,Valerie S. LeBleu,Tujin Shi,Suchitra Swaminathan,Yuan Luo,Dominique Missiakas,Glenn Randall,Alexis R. Demonbreun,Michael G. Ison,Raghu Kalluri,Deyu Fang,Huiping Liu +35 more
TL;DR: In this paper , an increase in circulating extracellular vesicles (EVs) that express ACE2 (evACE2) in plasma of COVID-19 patients was associated with severe pathogenesis.
A Cdh3-β-catenin-laminin signaling axis in a subset of breast tumor leader cells control leader cell polarization and directional collective migration
Priscilla Y. Hwang,Jairaj Mathur,Ya-Hui Cao,José António de Almeida,Jiayu Ye,Vasilios A. Morikis,Daphne Cornish,M. Clarke,Sheila A. Stewart,Amit Pathak,Gregory D. Longmore +10 more
TL;DR: In this paper , the authors developed 3D computational models, which hypothesize that leader cells need to generate high protrusive forces and overcome extracellular matrix resistance at the leading edge.
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A Cdh3-Lam332 signaling axis in a leader cell subpopulation controls protrusion dynamics and tumor organoid collective migration
Priscilla Y. Hwang,Jairaj Mathur,Ya-Hui Cao,José António de Almeida,Daphne Cornish,M. Clarke,Amit Pathak,Gregory D. Longmore +7 more
TL;DR: 3D computational models hypothesize that leader cells generate high protrusive forces and overcome extracellular matrix (ECM) resistance and identify and isolate a unique Cadherin-3 (Cdh3) positive leader cell subpopulation that is necessary and sufficient to lead migration.
Circulating ACE2-expressing extracellular vesicles block broad strains of SARS-CoV-2
Lamiaa El-Shennawy,Andrew D. Hoffmann,Nurmaa Dashzeveg,Kathleen M. McAndrews,P.J. Mehl,Daphne Cornish,Zihao Yu,Valerie Tokars,Vlad Nicolaescu,Anastasia Tomatsidou,Chengsheng Mao,Christopher Felicelli,Chia-Feng Tsai,Carolina Ostiguin,Yuzhi Jia,Lin Li,Kevin Furlong,Jan Wysocki,Xin Luo,Carolina F. Ruivo,Daniel Batlle,Thomas J. Hope,Yang Shen,Young Kwang Chae,Hui Zhang,Valerie S. LeBleu,Tujin Shi,Suchitra Swaminathan,Yuan Luo,Dominique Missiakas,Glenn Randall,Alexis R. Demonbreun,Michael G. Ison,Raghu Kalluri,Deyu Fang,Huiping Liu +35 more
TL;DR: In this paper , an increase in circulating extracellular vesicles (EVs) that express ACE2 (evACE2) in plasma of COVID-19 patients was associated with severe pathogenesis.