Daniel H. Conway
Drexel University
6 Papers
154 Citations
Daniel H. Conway is an academic researcher from Drexel University. The author has contributed to research in topics: Cellular differentiation & Odds ratio. The author has an hindex of 4, co-authored 5 publications.
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Papers
Prevalence of congenital anomalies in infants with in utero exposure to antiretrovirals.
Katherine M. Knapp,Susan B. Brogly,Daniel G. Muenz,Hans M. L. Spiegel,Daniel H. Conway,Gwendolyn B. Scott,Jeffrey T. Talbot,David Shapiro,Jennifer S. Read +8 more
TL;DR: The observed rate of congenital anomalies in this cohort is higher than previously reported for the general population, but it is consistent with rates observed in other recent studies of children born to human immunodeficiency virus–infected women.
Birth Defects Among Children Born to Human Immunodeficiency Virus-Infected Women Pediatric AIDS Clinical Trials Protocols 219 and 219C
Susan B. Brogly,Mark J. Abzug,D. Heather Watts,Coleen K. Cunningham,Paige L. Williams,James M. Oleske,Daniel H. Conway,Rhoda S. Sperling,Hans M. L. Spiegel,Russell B. Van Dyke +9 more
TL;DR: In this article, the authors used Logistic regression models to evaluate associations between first trimester in utero antiretroviral therapy exposure and birth defects, and found that the risk of birth defects was higher among children with first-trimester efavirenz exposure (5/32, 156%) versus children without first-term exposure (adjusted odds ratio [aOR] = 431 [95% CI: 156, 1186]), while a higher risk of heart defects was found.
Truncus arteriosus and other lethal internal anomalies in Goltz syndrome.
Xiang Y. Han,Sandy S. Wu,Daniel H. Conway,Bruce R. Pawel,Hope H. Punnett,Rick A. Martin,Jean-Pierre de Chadarévian +6 more
TL;DR: An infant girl of 36 weeks gestational age was found to have cardiovascular and other lethal internal anomalies in addition to characteristic external abnormalities of focal dermal hypoplasia (Goltz syndrome); such a combination of severe anomalies has not been reported previously in Goltz Syndrome.
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Humans with inherited MyD88 and IRAK-4 deficiencies are predisposed to hypoxemic COVID-19 pneumonia
Ana García-García,Rebeca Pérez de Diego,Carlos Flores,Darawan Rinchai,Jordi Solé-Violán,Angela Deyà-Martínez,Blanca García-Solis,José M. Lorenzo-Salazar,Elisa Hernández-Brito,Anna-Lisa Lanz,Leen Moens,Giorgia Bucciol,Mohamed A Almuqamam,Joseph B. Domachowske,Elena Colino,Juan Luis Santos-Perez,Francisco Marco,Claudio Pignata,Aziz Bousfiha,Stuart E. Turvey,S. Baur,Filomeen Haerynck,Javier Gonzalo Ocejo-Vinyals,Francisco Lendínez,Seraina Prader,Nora Naumann-Bartsch,Jana Pachlopnik Schmid,Catherine M. Biggs,Kyla J. Hildebrand,Alexandra Dreesman,Miguel Angel Cárdenes,Fatima Ailal,Ibtihal Benhsaien,Giuliana Giardino,Águeda Molina-Fuentes,Claudia Fortuny,Swetha Madhavarapu,Daniel H. Conway,Carolina Prando,Laire Schidlowski,María Teresa Martínez de Saavedra Álvarez,Rafael Alfaro,Felipe Rodríguez de Castro,Isabelle Meyts,Fabian Hauck,Anne Puel,Paul Bastard,Bertrand Boisson,Emmanuelle Jouanguy,Laurent Abel,Aurélie Cobat,Qian Zhang,Jean-Laurent Casanova,Laia Alsina,Carlos Rodríguez-Gallego +54 more
TL;DR: MyD88 and IRAK-4-deficient patients have a higher risk of hypoxemic COVID-19 pneumonia than individuals of similar age in the general population, due to impaired TLR7-dependent type I IFN production as mentioned in this paper .