Daniel Barson
University of Cambridge
7 Papers
Daniel Barson is an academic researcher from University of Cambridge. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 1, co-authored 1 publications.
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Papers
Astrocyte response to motor neuron injury promotes structural synaptic plasticity via STAT3-regulated TSP-1 expression
Giulia E. Tyzack,Sergey Sitnikov,Daniel Barson,Kerala L. Adams-Carr,Nike K. Lau,Jessica C. F. Kwok,Chao Zhao,Robin J.M. Franklin,Ragnhildur Thóra Káradóttir,James W. Fawcett,Andras Lakatos +10 more
TL;DR: It is demonstrated that TSP-1 is responsible for the remote AC-mediated recovery of excitatory synapses onto axotomized motor neurons in adult mice, providing new targets for neuroprotective therapies via optimizing AC-driven plasticity.
Why is everyone talking about brain state?
TL;DR: In this article , a parsimonious conceptualization of brain state as the fundamental building block of whole-brain activity offers a common framework to relate findings across scales and species, and examples of the diverse techniques commonly used to study brain states associated with physiology and higher-order cognitive processes.
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Rapid fluctuations in functional connectivity of cortical networks encode spontaneous behavior.
Hadas Benisty,Daniel Barson,Andrew H. Moberly,Sweyta Lohani,L. Tang,Ronald R. Coifman,Michael C Crair,Gal Mishne,Jessica A. Cardin,Michael J. Higley +9 more
TL;DR: Wide-field mesoscopic calcium imaging is used to monitor cortical dynamics in awake mice and an approach to quantify rapidly time-varying functional connectivity is developed, which provides insight into the relationship between neural signals and behavior.
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Hebbian instruction of axonal connectivity by endogenous correlated spontaneous activity
Naoyuki Matsumoto,Daniel Barson,Liang Liang,Michael C. Crair +3 more
TL;DR: Simultaneous in vivo imaging reveals that endogenous correlated spontaneous activity instructs axonal connectivity with subcellular precision, following Hebb's law, where synchronized activity promotes branch addition and desynchronized activity leads to elimination, refining neural circuits before sensory experience.
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In situ imaging of retinal calcium dynamics in awake animals
TL;DR: In this paper , the authors stabilize the eye to access in situ retinal dynamics with optical techniques in awake mice, and demonstrate that retinal activity is strongly modulated by movement through H1R-dependent histaminergic transmission in vivo, even at the amacrine cell level.
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