Da P Chen
6 Papers
Da P Chen is an academic researcher. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 2, co-authored 6 publications.
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Papers
Anti-DFS70 antibodies in systemic lupus erythematosus: Prevalence in a large Chinese cohort and an unexpected association with anti-dsDNA antibodies by a long-term follow-up
Y. Dai,Enling Li,Da P Chen,X. Niu,Zhiqing Wang,Liangjing Lu,Bing Zheng +6 more
TL;DR: The positive association of anti-DFS70 with anti-dsDNA and consistent dynamic variation between anti- DFS70 and anti-DSDNA during the follow-up suggested a potential relationship between anti the non-isolated anti-dfS70 in patients with SLE.
Anti-DFS70 Antibodies Are Associated With Proliferative Lupus Nephritis and Renal Pathological Activity
TL;DR: Anti-DFS70 antibodies were not associated with LN development in SLE patients but were associated with anti-dsDNA antibodies, proliferative LN, and renal AI, suggesting their potential to serve as a non-histological biomarker for LN subclass and activity status.
Local injection of micro-dose anti-interleukin-17A antibody for palmoplantar pustulosis: A real-world study.
TL;DR: This is the first study to evaluate the efficacy and safety of local injection of micro-dose ixekizumab for PPP in real clinical practice and a high proportion of patients rapidly achieved PPPASI 75, and maintained long-term efficacy with satisfactory safety.
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An Intravenous Immunoglobulin Scheme Successfully Preventing Recurrent CHB in Fetuses of Anti-SSA/SSB-Positive Mothers.
TL;DR: In this article , the authors investigated myocardial fibrosis assessed by T1 mapping in patients with apparently idiopathic premature ventricular complexes (PVCs), and to determine the relationship between this tissue biomarker and PVC features.
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Familial systemic lupus erythematosus associated with a loss-of-function mutation in TRPC6
TL;DR: In this article , the authors found a novel heterozygous mutation of the transient receptor potential channel 6 (TRPC6) gene, an NCF1ROS downstream calcium channel activated by diacylglycerol, and it acts as a lossoffunction mutation in an SLE family.