D. Powell
Harvard University
17 Papers
288 Citations
D. Powell is an academic researcher from Harvard University. The author has contributed to research in topics: Parathyroid hormone & Hormone. The author has an hindex of 13, co-authored 17 publications. Previous affiliations of D. Powell include Tokyo Medical and Dental University.
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Papers
Mullerian inhibiting substance ontogeny and its modulation by follicle-stimulating hormone in the rat testes
Tatsuo Kuroda,Mary M. Lee,Christopher M. Haqq,D. Powell,Thomas F. Manganaro,Patricia K. Donahoe +5 more
TL;DR: It is suggested that FSH may be a modulator of MIS production in neonatal testes and that the inhibitory effect on testicular MIS production may require continued FSH exposure.
81
Preoperative localization of abnormal parathyroid tissue. Cumulative experience with venous sampling and arteriography.
John P. Bilezikian,John L. Doppman,Peter M. Shimkin,D. Powell,Samuel A. Wells,D.A. Heath,Alfred S. Ketcham,J. M. Monchik,Lawrence E. Mallette,John T. Potts,Gerald D. Aurbach +10 more
TL;DR: The results indicate that selective venous sampling and radioimmunoassay of parathyroid hormone are useful preoperative adjuncts to surgical exploration especially in patients who have had previous neck surgery.
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•Journal Article
Recombinant human mullerian inhibiting substance inhibits human ocular melanoma cell lines in vitro and in vivo.
TL;DR: The responsiveness of ocular melanoma to MIS broadens the spectrum of tumors that might be treated with MIS and suggests further investigation of other neural crest tumors.
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Dextran-charcoal and dioxane phase separation methods in the radioimmunoassays for parathyroid hormone and calcitonin.
TL;DR: Characterization of variables affecting bound radioactivity, and careful standardization of the conditions of phase separation, indicate that dioxane precipitation is a satisfactory method for the calcitonln immunoassay while dextran-charcoal adsorption provides the most reliable results in the parathyroid hormone immunoASSay.
24
Müllerian inhibiting substance blocks epidermal growth factor receptor phosphorylation in fetal rat lung membranes.
Elizabeth A. Catlin,Neal D. Uitvlugt,Patricia K. Donahoe,D. Powell,Masando Hayashi,David T. MacLaughlin +5 more
TL;DR: Electron microscopy of rhMIS-exposed lung showed decreased lamellar bodies (LB) in both male alveolar spaces and female parenchyma, and, unexpectedly, increased numbers in female alveoli.
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