D. Laubitz
Polish Academy of Sciences
33 Papers
159 Citations
D. Laubitz is an academic researcher from Polish Academy of Sciences. The author has contributed to research in topics: Biology & Internal medicine. The author has an hindex of 13, co-authored 25 publications. Previous affiliations of D. Laubitz include Lund University.
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Papers
Sodium-butyrate as a growth promoter in milk replacer formula for young calves.
Paul Guilloteau,Romuald Zabielski,J C David,J.W. Blum,Jean Morisset,M Biernat,J. Woliński,D. Laubitz,Y Hamon +8 more
TL;DR: Beneficial effects of Na-butyrate on maturation of gastrointestinal functions were shown in milk-fed calves and may be applied to young mammals of other species.
148
•Journal Article
Effect of sodium butyrate on the small intestine development in neonatal piglets fed [correction of feed] by artificial sow.
TL;DR: It is suggested that supplementation with Na-butyrate may enhance the development of jejunal and ileal mucosa in formula-fed piglets.
89
Reduced colonic microbial diversity is associated with colitis in NHE3-deficient mice
Claire B. Larmonier,D. Laubitz,Faihza M. Hill,Kareem W. Shehab,Leszek Lipinski,Monica T. Midura-Kiela,Rita-Marie T. McFadden,Rajalakshmy Ramalingam,Kareem Hassan,Marcin Gołębiewski,Marcin Gołębiewski,David G. Besselsen,Fayez K. Ghishan,Pawel R. Kiela +13 more
TL;DR: The gut microbiome plays a prominent role in the pathogenesis of colitis in N HE3(-/-) mice, and, reciprocally, NHE3 also plays a critical role in shaping the gut microbiota.
85
•Journal Article
Effect of sodium butyrate on the small intestine development in neonatal piglets feed by artificial sow
TL;DR: In this article, the effect of milk formula supplementation with Na-butyrate on the gut mucosa in neonatal piglets was investigated and the results suggest that supplementing with Nabutyrates may enhance the development of jejunal and ileal mucosa.
NHE3 modulates the severity of colitis in IL-10-deficient mice
Claire B. Larmonier,D. Laubitz,Robert D. Thurston,Alaxis L Bucknam,Faihza M. Hill,Monica T. Midura-Kiela,Rajalakshmy Ramalingam,Pawel R. Kiela,Fayez K. Ghishan +8 more
TL;DR: Chronic NHE3 inhibition or underexpression observed in IBD may contribute to the pathogenesis of IBD by influencing the extent of the epithelial barrier defect and affect the ultimate degree of inflammation.