D. Feuvray
University of Paris
31 Papers
709 Citations
D. Feuvray is an academic researcher from University of Paris. The author has contributed to research in topics: Intracellular pH & Diabetes mellitus. The author has an hindex of 18, co-authored 31 publications. Previous affiliations of D. Feuvray include St Thomas' Hospital & University of Tromsø.
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Papers
Protection of the ischemic myocardium. Ultrastructural, enzymatic, and functional assessment of the efficacy of various cardioplegic infusates.
P. Jynge,P. Jynge,P. Jynge,D.J. Hearse,D.J. Hearse,D.J. Hearse,J. De Leiris,J. De Leiris,J. De Leiris,D. Feuvray,D. Feuvray,D. Feuvray,M. V. Braimbridge,M. V. Braimbridge,M. V. Braimbridge +14 more
TL;DR: Assessment of the efficacy of three infusates in a rat heart model of cardiopulmonary bypass and ischemic cardiac arrest indicates that the St. Thomas' Hospital solution is an effective protective agent under all conditions studied but the Bretschneider solution is effective only under hypothermic conditions and the Kirsch solution is ineffective and may exacerbate tissue injury.
157
Controversies on the sensitivity of the diabetic heart to ischemic injury : the sensitivity of the diabetic heart to ischemic injury is decreased
D. Feuvray,Gary D. Lopaschuk +1 more
TL;DR: It is suggested that although impaired glucose metabolism in the diabetic heart contributes to injury in hypoxic hearts or in hearts subjected to low-flow ischemia, diabetes-induced decreases in glycolysis can actually be beneficial to the diabeticheart during and following a severe ischemic episode.
145
Changes in intracellular sodium and pH during ischaemia-reperfusion are attenuated by trimetazidine. Comparison between low- and zero-flow ischaemia.
Houda El Banani,Monique Bernard,D. Baetz,Emmanuel Cabanes,Patrick J. Cozzone,Arnaud Lucien,D. Feuvray +6 more
TL;DR: In both LFI and TI, TMZ-induced attenuation of ionic imbalance was associated with a significantly improved recovery of ventricular function on reperfusion, as assessed by a lower increase in diastolic pressure and an increased recovery of developed pressure.
Intracellular pH and role of Na+/H+ exchange during ischaemia and reperfusion of normal and diabetic rat hearts.
TL;DR: Comparison of data from diabetic rat hearts with reduced activity of the Na+/H+ exchange process v normal hearts with pharmacological block of the exchanger provide support for a critical role of theNa+/ H+ exchanger in the initial stage of reperfusion.
103
Enhanced activity of the myocardial Na+/H+ exchanger contributes to left ventricular hypertrophy in the Goto-Kakizaki rat model of type 2 diabetes: critical role of Akt.
TL;DR: These findings indicate that activation of the Akt pathway represents a likely mechanism mediating the hypertrophic effect of increased NHE1 activity in the GK model of type 2 diabetes.