Cyntia Pecli
Federal University of Rio de Janeiro
4 Papers
Cyntia Pecli is an academic researcher from Federal University of Rio de Janeiro. The author has contributed to research in topics: CCL17 & Cytokine. The author has an hindex of 3, co-authored 3 publications.
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Papers
Lack of galectin-3 speeds Wallerian degeneration by altering TLR and pro-inflammatory cytokine expressions in injured sciatic nerve.
Bruno Siqueira Mietto,Sofia Jürgensen,Lucineia Alves,Cyntia Pecli,Marcelo Sampaio Narciso,Iranaia Assunção-Miranda,Déa Maria Serra Villa-Verde,Flavia Regina Souza Lima,João R. L. Menezes,Claudia F. Benjamim,Marcelo T. Bozza,Ana Maria Blanco Martinez +11 more
TL;DR: Data indicate that the lack of Gal‐3 results in an augmented inflammatory profile that involves the TLR–cytokine pathway, and increases the phagocytic capacity of Schwann cells and macrophages, which ultimately contributes to speeding the course of WD.
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Role of Chemokine Receptor CCR4 and Regulatory T Cells in Wound Healing of Diabetic Mice
Janaína F. Barros,Ingrid Waclawiak,Cyntia Pecli,Paula A. Borges,Janaína L. Georgii,Erivan Schnaider Ramos-Junior,Claudio Canetti,Tristan Courau,David Klatzmann,David Klatzmann,Steven L. Kunkel,Carmen Penido,Fábio B. Canto,Fábio B. Canto,Claudia F. Benjamim +14 more
TL;DR: The data suggest that the receptor CCR4 and regulatory T cells negatively affect wound healing in diabetic mice.
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Pleiotropic antifibrotic actions of aspirin-triggered resolvin D1 in the lungs
Rafael F. Guilherme,José Bruno Nunes Ferreira Silva,Ingrid Waclawiack,Vanderlei da Silva Fraga-Junior,Thaís O. Nogueira,Cyntia Pecli,Carlla Assis Araujo-Silva,Nathalia Santos Magalhães,Felipe S. Lemos,Carlos A. Bulant,Pablo J. Blanco,Rafaela Rangel Serra,Erik Svensjö,Julio Scharfstein,João Alfredo Moraes,Claudio Canetti,Claudia F. Benjamim +16 more
TL;DR: In this paper , the authors used a mouse model of bleomycin (BLM)-induced lung fibrosis, and injected aspirin-triggered resolvin D1 (7S,8R,17R-trihydroxy-4Z,9E,11E,13Z,15E19Z-docosahexaenoic acid) 7 and 10 days after BLM (intracheal) challenge and samples were two weeks later.
CCR4 Controls the Suppressive Effects of Regulatory T Cells on Early and Late Events during Severe Sepsis
Raphael Molinaro,Cyntia Pecli,Rafael F. Guilherme,José C. Alves-Filho,Fernando Q. Cunha,Claudio Canetti,Steven L. Kunkel,Marcelo T. Bozza,Claudia F. Benjamim +8 more
TL;DR: Data show that CCR4 is involved in immunosuppression after severe sepsis and suggest that C CR4+ Tregs negatively modulate the short and long-term immune responses.