Cong Si Tran
4 Papers
Cong Si Tran is an academic researcher. The author has contributed to research in topics: Biology & Cancer. The author has co-authored 2 publications.
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Papers
Comparison of HAV and HCV infections in vivo and in vitro reveals distinct patterns of innate immune evasion and activation
Ombretta Colasanti,Rani Burm,Hao En Huang,Tobias Riedl,Jannik Traut,Nadine Gillich,Teng-Feng Li,Laura Corneillie,Suzanne Faure-Dupuy,Oliver Grünvogel,Danijela Heide,Ji-Young Lee,Cong Si Tran,Uta Merle,Maria Chironna,Florian W. R. Vondran,Katharina Esser-Nobis,Marco Binder,Ralf Bartenschlager,Mathias F. Heikenwälder,Philip Meuleman,Volker Lohmann +21 more
TL;DR: In this article, the authors investigated the mechanisms of HAV and hepatitis C virus (HCV) induction and counteraction by HAV proteases in vivo and in vitro and found that HAV 3ABC and 3CD proteases do not abolish the host innate immune response.
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Corrigendum to "Comparison of HAV and HCV infections in vivo and in vitro reveals distinct patterns of innate immune evasion and activation" [J Hepatol (2023) 645-656].
Ombretta Colasanti,Rani Burm,Hao-En Huang,Tobias Riedl,Jannik Traut,Nadine Gillich,Teng-Feng Li,Laura Corneillie,Suzanne Faure-Dupuy,Oliver Grünvogel,Danijela Heide,Ji-Young Lee,Cong Si Tran,Uta Merle,Maria Chironna,Florian F W Vondran,Katharina Esser-Nobis,Marco Binder,Ralf Bartenschlager,Mathias Heikenwälder,Philip Meuleman,Volker Lohmann +21 more
TL;DR: A corrigendum corrects an error in Fig. 4G of a study comparing HAV and HCV infections, where HAV's 3ABC protein size was mislabelled as 75 kDa instead of 38 kDa, affecting understanding of immune evasion mechanisms.
Phosphatidylinositol 4-kinase III alpha governs cytoskeletal organization for invasiveness of liver cancer cells.
Cong Si Tran,Julia Kersten,Jingyi Yan,Marco Breinig,Thorben Huth,Tanja Poth,Ombretta Colasanti,Tobias Riedl,Suzanne Faure-Dupuy,Stefan Diehl,Lieven Verhoye,Teng-Feng Li,Marit Lingemann,Philipp Schult,Gustaf Ahlén,Lars Frelin,Florian Kühnel,F. W. Vondran,Kai Breuhahn,Philip Meuleman,Mathias Heikenwälder,Peter Schirmacher,R. Bartenschlager,Vibor Laketa,Stephanie Roessler,Darjus F. Tschaharganeh,Matti Sällberg,Volker Lohmann +27 more
TL;DR: Mechanistic insight is provided into the contribution of PI4KIIIα and HCV to progression of liver cancer and promising targets for therapeutic intervention are identified.
Phosphatidylinositol 4-kinase III alpha governs cytoskeletal organization for invasiveness of liver cancer cells
Cong Si Tran,Julia Kersten,Marco Breinig,Jingyi Yan,Tanja Poth,Ombretta Colasanti,Tobias Riedl,Suzanne Faure-Dupuy,Stefan Diehl,Lieven Verhoye,Teng-Feng Li,Philipp Schult,Gustaf Ahlén,Lars Frelin,Florian Kühnel,Kai Breuhahn,Florian W. R. Vondran,Philip Meuleman,Peter Schirmacher,Matti Sällberg,Ralf Bartenschlager,Vibor Laketa,Darjus F. Tschaharganeh,Volker Lohmann +23 more
TL;DR: In this article , the impact of liver cancer on hepatocellular carcinoma (HCC) was examined by using hepatitis C virus (HCV) infection or overexpression of its nonstructural protein.