Chuang Wei Wang
Chang Gung University
6 Papers
Chuang Wei Wang is an academic researcher from Chang Gung University. The author has contributed to research in topics: Medicine & Wnt signaling pathway. The author has an hindex of 4, co-authored 6 publications. Previous affiliations of Chuang Wei Wang include Memorial Hospital of South Bend.
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Papers
Whole genome sequencing identifies genetic variants associated with co-trimoxazole hypersensitivity in Asians.
Chuang Wei Wang,Wichittra Tassaneeyakul,Chun-Bing Chen,Wei-Ti Chen,Yu Chuan Teng,Cheng-Yang Huang,Chonlaphat Sukasem,Chun-Wei Lu,Yun-Shien Lee,Siew Eng Choon,Nontaya Nakkam,Rosaline Chung-Yee Hui,Yen-Hua Huang,Ya Ching Chang,Yang Yu Wei Lin,Chee-Jen Chang,Tsu Man Chiu,Wasun Chantratita,Parinya Konyoung,Chaw Ning Lee,Jettanong Klaewsongkram,Ticha Rerkpattanapipat,Warayuwadee Amornpinyo,Niwat Saksit,Pawinee Rerknimitr,Yu-Huei Huang,Shang Hung Lin,Chao Kai Hsu,Cheng Chi Chan,Yu Jr Lin,Shuen-Iu Hung,Wen-Hung Chung +31 more
TL;DR: This study identified HLA-B*13:01 is an important genetic factor associated with co-trimoxazole-induced SCAR in Asians and identified a strong HLA association in the cases from Thailand and Malaysia.
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Treatment of epidermal growth factor receptor inhibitor-induced severe paronychia with pyogenic granuloma-like lesions with topical betaxolol: an open-label observation study.
Chi Feng Yen,Chi Feng Yen,Chao Kai Hsu,Hsing San Yang,Chaw Ning Lee,Ching-Chi Chi,Ching-Chi Chi,Wen-Hung Chung,Wen-Hung Chung,Chih Liang Wang,Jong Hwei Su Pang,Jong Hwei Su Pang,Chuang Wei Wang,Chuang Wei Wang,Yu Shien Ko,Chun-Wei Lu,Chun-Wei Lu +16 more
TL;DR: The aim of this study is to find an effective, pain‐relieving, and noninvasive treatment for patients with severe paronychia induced by EGFR inhibitors.
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Attenuation of Wnt/β-catenin signaling in patients with Stevens-Johnson syndrome and toxic epidermal necrolysis.
Chun-Bing Chen,Wan-Chun Chang,Ming Ying Wu,Ming Ying Wu,Tzu Yang Kao,Ying Wen Wang,Chuang Wei Wang,Chuang Wei Wang,Chi Ju Chen,Wen-Hung Chung,Shih-Chi Su +10 more
TL;DR: Downregulation of Wnt signaling components, including T cell factor 1/lymphoid enhancer binding factor 1 (LEF-1) transcription factors, in SJS/TEN patients are shown, suggesting that canonical Wnt signaled is regulated during cytotoxic T cell responses in S JS/T EN.
The Medication Risk of Stevens–Johnson Syndrome and Toxic Epidermal Necrolysis in Asians: The Major Drug Causality and Comparison With the US FDA Label
Yu-Hsin Wang,Chun-Bing Chen,Wichittra Tassaneeyakul,Yoshiro Saito,Michiko Aihara,Siew Eng Choon,Haur Yueh Lee,Mimi Mee Chang,Francisca D Roa,Cheng-Wei Wu,Jing Zhang,Nontaya Nakkam,Parinya Konyoung,Yoshimi Okamoto-Uchida,Christina Man-Tung Cheung,Jin-wen Huang,Chao Ji,Bo Cheng,Rosaline Chung-Yee Hui,Rosaline Chung-Yee Hui,Chia-Yu Chu,Yi-Ju Chen,Ching-Ying Wu,Chao Kai Hsu,Tsu-Man Chiu,Yu-Huei Huang,Yu-Huei Huang,Chun-Wei Lu,Chin-Yi Yang,Chin-Yi Yang,Yi-Ting Lin,Yi-Ting Lin,Min-Hui Chi,Min-Hui Chi,Hsin-Chun Ho,Hsin-Chun Ho,Jing-Yi Lin,Jing-Yi Lin,Chih-Hsun Yang,Chih-Hsun Yang,Ya-Ching Chang,Ya-Ching Chang,Shih-Chi Su,Chuang Wei Wang,Wen-Lang Fan,Shuen-Iu Hung,Wen-Hung Chung +46 more
TL;DR: Only one acetaminophen‐induced SJS was identified, while several medications labeled as carrying a risk of SJS/TEN by the FDA were not found to have caused any of the cases in the Asian countries investigated in this study.
Delayed-type hypersensitivity reactions induced by proton pump inhibitors: A clinical and in vitro T-cell reactivity study.
Chien-Yio Lin,Chuang Wei Wang,C.-Y. R. Hui,Ya-Ching Chang,C.-H. Yang,Chi-Yuan Cheng,W.-W. Chen,W.-M. Ke,Wen-Hung Chung +8 more
TL;DR: A study of a large series of PPI-related DHR patients, followed up their tolerability to alternative anti‐ulcer agents, and investigated the T‐cell reactivity to PPI in PPI‐related D HR patients.