Christopher M. Johnson
Mayo Clinic
26 Papers
259 Citations
Christopher M. Johnson is an academic researcher from Mayo Clinic. The author has contributed to research in topics: Autosomal dominant polycystic kidney disease & Kidney. The author has an hindex of 14, co-authored 26 publications. Previous affiliations of Christopher M. Johnson include Karolinska Institutet.
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Papers
Renal stone epidemiology: A 25-year study in Rochester, Minnesota
TL;DR: The first description of the incidence and recurrence rates for symptomatic noninfected renal stones in a well-defined population of Rochester, Minnesota, between 1950 and the end of 1974 is described.
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Synthesis of renin by tubulocystic epithelium in autosomal-dominant polycystic kidney disease.
Vicente E. Torres,Kathleen A. Donovan,G Scicli,Keith E. Holley,Stephen N. Thibodeau,Oscar A. Carretero,Tadashi Inagami,James A. McAteer,Christopher M. Johnson +8 more
TL;DR: Results indicate that the tubulocystic epithelium has the potential to synthesize renin, and Elevated levels of active renin in renal cysts may be linked to the pathogenesis of hypertension in ADPKD.
146
Porcine cardiac valvular subendothelial cells in culture: cell isolation and growth characteristics.
TL;DR: It is concluded that valve subendothelial cells show features that distinguish them from other cultured mesenchymal cells, and that this culture system will be useful for studies of the cellular basis of valvular heart disease.
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Effect of Inhibition of Converting Enzyme on Renal Hemodynamics and Sodium Management in Polycystic Kidney Disease
Vicente E. Torres,David M. Wilson,John C. Burnett,Christopher M. Johnson,Kenneth P. Offord +4 more
- 01 Oct 1991
TL;DR: The results suggest that the renal renin-angiotensin system plays a central role in the alterations in renal hemodynamics and sodium management associated with the development of hypertension in ADPKD.
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Presence of antibodies in the sera of patients with Graves' disease recognizing a 23 kilodalton fibroblast protein.
TL;DR: The results suggest the possibility that antibodies directed against this fibroblast antigen may be related to the development of other thyroid disorders or autoimmune diseases.
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