Chris Galanos
Max Planck Society
176 Papers
4.3K Citations
Chris Galanos is an academic researcher from Max Planck Society. The author has contributed to research in topics: Lipid A & Lipopolysaccharide. The author has an hindex of 63, co-authored 175 publications. Previous affiliations of Chris Galanos include University of Texas Southwestern Medical Center.
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Papers
TNF-α hyper-responses to Gram-negative and Gram-positive bacteria in Propionibacterium acnes primed or Salmonella typhimurium infected mice:
TL;DR: Although sensitization to LPS and killed Listeria monocytogenes is exclusively IFN-γ-dependent, an IFN -γ-independent, moderate sensitized to certain TNF-α-inducing constituents in bacteria may develop in parallel.
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Differential clearance and induction of host responses by various administered or released lipopolysaccharides.
Ryoichi Hasunuma,Hiroyuki Morita,Shigenori Tanaka,Roland Ryll,Marina A. Freudenberg,Chris Galanos,Yoshio Kumazawa +6 more
TL;DR: Results indicate that continuously higher levels of plasma LPS followed by stronger host responses occur during infection and suggest that these differences between LPS-administered and infected mice should be taken into consideration when analyzing host responses induced by LPS.
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Identification and percentage frequency of isolated non-parenchymal liver cells (NPLC) in the mouse
Nikolaus Freudenberg,J. Schalk,Chris Galanos,T. H. Katschinski,O. Datz,U. Pein,Marina A. Freudenberg +6 more
TL;DR: The results show that, apart from the more familiar varieties of NPLC, two groups of cells exist in the liver which have not yet been fully identified and in which the immunocompetent cells predominate numerically.
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Beneficial or Deleterious Effects of a Preexisting Hypersensitivity to Bacterial Components on the Course and Outcome of Infection
TL;DR: Priming with heat-killed Propionibacterium acnes enhances the sensitivity of mice to lipopolysaccharide (LPS) and other biologically active bacterial components and has protective and deleterious effects on a subsequent serovar Typhimurium infection.
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Dietary testosterone suppresses protective responsiveness to Plasmodium chabaudi malaria.
Horst Mossmann,W. Peter M. Benten,Chris Galanos,Marina A. Freudenberg,W. Nikolaus Kühn-Velten,Hans Reinauer,Frank Wunderlich +6 more
TL;DR: The data indicate that the method of oral administration of 17 alpha-methyl-testosterone raises circulating testosterone to levels that impair protective immune responses to P. chabaudi malaria.
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