Chris Galanos
Max Planck Society
176 Papers
4.3K Citations
Chris Galanos is an academic researcher from Max Planck Society. The author has contributed to research in topics: Lipid A & Lipopolysaccharide. The author has an hindex of 63, co-authored 175 publications. Previous affiliations of Chris Galanos include University of Texas Southwestern Medical Center.
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Papers
Influence of EDTA and heparin on lipopolysaccharide binding and cell activation, evaluated at single-cell level in whole blood
Milena Karina Coló Brunialti,Esper G. Kallas,Marina A. Freudenberg,Chris Galanos,Reinaldo Salomão +4 more
TL;DR: In this paper, the authors evaluated LPS binding and cell activation in WB by using flow cytometry, and the influence of heparin or EDTA as anticoagulants was also addressed.
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A method to detect 2-keto-3-deoxyoctanat and related compounds on pherograms and chromatograms
Helmut Brade,Chris Galanos +1 more
TL;DR: A variation of the thiobarbituric acid spray reagent to detect 2-keto-3-deoxyoctanat (KDO), KDO derivatives, 2- deoxysugars, and N-acetylneuraminic acid on pherograms and chromatograms is described.
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Experimental lipid A-induced nephritis in the dog
M Westenfelder,Chris Galanos +1 more
TL;DR: The effect of radioactive lipid A, obtained from Escherichia coli, on the kidney of adult dogs and puppies was investigated in this article, where the intensity and duration of the kidney response coorelated well with the lipid A dose administered.
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Comparison of the capacity of two lipid A precursor molecules to express the local Shwartzman phenomenon.
TL;DR: The results indicate that the additional presence of at least one fatty acid either as such or as an acyloxyacyl residue is a prerequisite for the ability of the molecule to induce the local Shwartzman phenomenon.
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•Journal Article
Tumour Biology: tumour-associated inflammation versus antitumor immunity.
Boris Müller-Hübenthal,Marc Azemar,D. Lorenzen,Michael Huber,Marina A. Freudenberg,Chris Galanos,Clemens Unger,Bernd Hildenbrand +7 more
TL;DR: The latest research results suggest that tumour-infiltrating leukocytes and the intra-tumoural cytokine environment play a central role in both the genesis and development of cancer.
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