Chen Peng
Tongji University
18 Papers
31 Citations
Chen Peng is an academic researcher from Tongji University. The author has contributed to research in topics: Medicine & Psoriasis. The author has an hindex of 2, co-authored 10 publications.
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Papers
Psoriasis comorbidities
TL;DR: This review summarizes psoriasis-associated comorbidities that have been reported in studies, and analyzes the possible co-pathogenesis of these diseases and Psoriasis.
20
Epidemiological Variations in the Global Burden of Psoriasis, an Analysis With Trends From 1990 to 2017.
TL;DR: In this paper, the authors evaluated trends and variations in the global health burden of psoriasis using disability-adjusted life years (DALYs) from the Global Burden of Disease Study.
Association between systemic immune inflammation index, systemic inflammation response index and adult psoriasis: evidence from NHANES
Rui Ma,Lian Cui,Jiangluyi Cai,Nan Yang,Yuanyuan Wang,Qianyu Chen,Wenjuan Chen,Chen Peng,Hui Qin,Yangfeng Ding,Xin Wang,Yuling Shi +11 more
TL;DR: SII and SIRI are positively associated with psoriasis. The association is non-linear and stronger in females, obese individuals, people with type 2 diabetes, and those without hypercholesterolemia.
15
Measurement properties of the Patient Global Assessment numerical rating scale in moderate-to-severe psoriasis.
Ning Yu,Chen Peng,Jing Zhou,Junying Gu,Jinhua Xu,Xia Li,Xinling Bi,Hui Deng,Xin Li,Yangfeng Ding,Yuling Shi +10 more
TL;DR: In this article , a single-question 11-point Patient Global Assessment (PtGA) numeric rating scale (NRS) was validated in patients with plaque psoriasis in a prospective, multicenter, and observational registry.
11
Circulating Metabolomic Signature in Generalized Pustular Psoriasis Blunts Monocyte Hyperinflammation by Triggering Amino Acid Response.
Ning Yu,Chen Peng,Wenjuan Chen,Ziwen Sun,Jianfeng Zheng,Shujie Zhang,Yangfeng Ding,Yuling Shi +7 more
TL;DR: In this article, a metabolomic, functional, and mechanistic analysis of generalized pustular psoriasis (GPP) patients was performed, and it was shown that decreased serum amino acids in GPP blunted the innate immune response in blood monocytes through AAR pathway, serving as a feedback mechanism preventing excessive inflammation.