Chad D. Geringer
Eli Lilly and Company
5 Papers
23 Citations
Chad D. Geringer is an academic researcher from Eli Lilly and Company. The author has contributed to research in topics: Biology & Gene chip analysis. The author has an hindex of 4, co-authored 5 publications.
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Papers
Genome of the bacterium Streptococcus pneumoniae strain R6.
JoAnn Hoskins,William E. Alborn,Jeffrey S. Arnold,Larry C. Blaszczak,Stanley Gene Burgett,Bradley S. DeHoff,Shawn T. Estrem,Lori Fritz,Dong-Jing Fu,Wendy Fuller,Chad D. Geringer,Raymond Gilmour,Jennifer S. Glass,Hamid Khoja,Angelika R. Kraft,Robert E. Lagace,Donald J. LeBlanc,Linda N. Lee,Elliot J. Lefkowitz,Jin Lu,Patti Matsushima,Scott M. McAhren,Margaret A. Mchenney,Kevin McLeaster,Christopher W. Mundy,Thalia I. Nicas,Franklin H. Norris,MaryJeanne O'Gara,Robert B. Peery,Gregory T. Robertson,Pamela Kay Rockey,Pei-Ming Sun,Malcolm E. Winkler,Yong Yang,Michelle Young-Bellido,Genshi Zhao,Christopher A. Zook,Richard H. Baltz,S. Richard Jaskunas,Paul Robert Rosteck,Paul L. Skatrud,John I. Glass +41 more
TL;DR: In the analysis of the genome, a large number of new uncharacterized genes predicted to encode proteins that either reside on the surface of the cell or are secreted are identified and there may be new targets for vaccine and antibiotic development.
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Aldosterone stimulates angiotensin-converting enzyme expression and activity in rat neonatal cardiac myocytes.
Jian Wang,Lan Yu,Patricia J. Solenberg,Lawrence M. Gelbert,Chad D. Geringer,Mitchell I. Steinberg +5 more
TL;DR: ACE gene expression may be a target for mineralocorticoid receptors in the myocardium, supporting the notion that at least some of the known adverse effects of aldosterone on the myCardium are mediated by increased angiotensin II.
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Assessing the Variability in GeneChip® Data
Shuguang Huang,Hui-Rong Qian,Chad D. Geringer,Christy Love,Lawrence M. Gelbert,Kerry G. Bemis +5 more
TL;DR: This paper introduces statistical methods to assess the variability of Affymetrix GeneChip® data due to randomness and finds that the biological and chip variation are roughly comparable.
Analysis of regulator of G‐protein signaling‐2 (RGS‐2) expression and function in osteoblastic cells
TL;DR: The data suggest that PTH treatment results in a direct transcriptional stimulation of RGS‐2 that in turn may play a role in modulating the duration/intensity of PTH receptor signaling.