Cameron C. Yin
University of Texas MD Anderson Cancer Center
14 Papers
62 Citations
Cameron C. Yin is an academic researcher from University of Texas MD Anderson Cancer Center. The author has contributed to research in topics: Myeloid leukemia & Fluorescence in situ hybridization. The author has an hindex of 8, co-authored 14 publications.
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Papers
inv(16)(p13q22) in chronic myelogenous leukemia in blast phase: a clinicopathologic, cytogenetic, and molecular study of five cases.
Mihai Merzianu,L. Jeffrey Medeiros,Jorge E. Cortes,Cameron C. Yin,Pei Lin,Dan Jones,Armand B. Glassman,Hagop M. Kantarjian,Yang Huh +8 more
TL;DR: 5 patients with CML in blast phase (CML-BP) in which t(9;22) and inv(16)(p13q22) were identified by conventional cytogenetics, with confirmation of BCR-ABL and CBFss-MYH11 by fluorescence in situ hybridization are described.
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Characteristics and outcomes of patients with V299L BCR-ABL kinase domain mutation after therapy with tyrosine kinase inhibitors.
Elias Jabbour,Van K. Morris,Hagop M. Kantarjian,Cameron C. Yin,Elizabeth M. Burton,Jorge E. Cortes +5 more
TL;DR: Point mutations of the Bcr-Abl kinase domain (KD) are the most frequently identified mechanism of resistance in patients with chronic myeloid leukemia (CML) who fail tyrosine kinase inhibitor (TKI) therapy.
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IGK with conserved IGKV/IGKJ repertoire is expressed in acute myeloid leukemia and promotes leukemic cell migration
Chong Wang,Miaoran Xia,Miaoran Xia,Xiaoping Sun,Zhiqiao He,Fanlei Hu,Lei Chen,Carlos E. Bueso-Ramos,Xiaoyan Qiu,Xiaoyan Qiu,Cameron C. Yin +10 more
TL;DR: IGK is expressed in myeloblasts and mature myeloid cells from patients with non-hematopoietic neoplasms, and is involved in cell migration, suggesting that myeloids cells-derived IgK may have a role in leukemogenesis and may serve as a novel tumor marker for monitoring minimal residual disease and developing target therapy.
Aberrant EVI1 expression in acute myeloid leukemias associated with the t(3;8)(q26;q24)
Patrick A. Lennon,Lynne V. Abruzzo,L. Jeffrey Medeiros,Candy C. Cromwell,Xiang Zhang,Cameron C. Yin,Steven M. Kornblau,Marina Konopieva,Pei Lin +8 more
TL;DR: It is demonstrated that the t(3;8)(q26;q24) results in deregulated EVI1 expression, similar to other balanced or unbalanced chromosomal translocations involving chromosome 3q26.
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A genome-wide association study identifies WT1 variant with better response to 5-fluorouracil, pirarubicin and cyclophosphamide neoadjuvant chemotherapy in breast cancer patients
Lina Wu,Lu Yao,Hong Zhang,Tao Ouyang,Jinfeng Li,Tianfeng Wang,Zhaoqing Fan,Tie Fan,Benyao Lin,Cameron C. Yin,Yuntao Xie +10 more
TL;DR: Results suggest that WT1, located in the WT1 gene, may be a potential target of anthracycline-based neoadjuvant therapy for breast cancer.
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