C. Cristofre Martin
University of Ottawa
17 Papers
329 Citations
C. Cristofre Martin is an academic researcher from University of Ottawa. The author has contributed to research in topics: Zebrafish & Biology. The author has an hindex of 14, co-authored 17 publications. Previous affiliations of C. Cristofre Martin include University of Manitoba & University of Saskatchewan.
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Papers
A role for DNA methylation in gastrulation and somite patterning.
TL;DR: It is proposed that DNA methylation is required for normal gastrulation and subsequent patterning of the dorsal mesoderm in zebrafish embryos treated with 5-azacytidine and 5-aza-2-deoxycytidine.
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Disruption of zebrafish somite development by pharmacologic inhibition of Hsp90.
Zsolt Lele,Steven D. Hartson,C. Cristofre Martin,Luke Whitesell,Robert L. Matts,Patrick H. Krone +5 more
TL;DR: The data are consistent with there being a temporal and spatial requirement for Hsp90 function within somitic cells which is necessary for the formation of eng-2-expressing muscle pioneers and possibly other striated muscle fiber types.
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Histone deacetylase 1 (HDAC‐1) required for the normal formation of craniofacial cartilage and pectoral fins of the zebrafish
TL;DR: It is shown that histone deacetylases interact with nucleosomes to facilitate the formation of transcriptionally repressed chromatin, and that hdac‐1 is expressed throughout embryonic development of the zebrafish.
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Genotype-specific modifiers of transgene methylation and expression in the zebrafish, Danio rerio.
TL;DR: It is demonstrated that modifiers that act in a similar fashion to those identified in mice also exist in a non-mammalian vertebrate, the zebrafish Danio rerio, and this data suggest evolutionary conservation of the modifiers, at least within vertebrates, and imply that they and their actions are important in normal vertebrate development.
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Parent‐of‐origin specific effects on the methylation of a transgene in the zebrafish, Danio rerio
TL;DR: The inheritance of a transgene locus in the zebrafish, Daniorerio, is examined and it is demonstrated that its methylation is protected by the sex of the parent contributing the allele, identical to imprinting as seen in mammals.
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