Bo Ding
4 Papers
Bo Ding is an academic researcher. The author has contributed to research in topics: Medicine & Cancer. The author has an hindex of 4, co-authored 4 publications.
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Papers
Inflammatory cell-derived CXCL3 promotes pancreatic cancer metastasis through a novel myofibroblast-hijacked cancer escape mechanism.
Xiaoting Sun,Xingkang He,Yin Zhang,Kayoko Hosaka,Patrik Andersson,Jing Wu,Jieyu Wu,Xu Jing,Qiqiao Du,Xiaoli Hui,Bo Ding,Ziheng Guo,An Hong,Xuan Liu,Yan Wang,Qing Ji,Rudi Beyaert,Yunlong Yang,Qi Li,Yihai Cao +19 more
TL;DR: In this paper, the role of the IL-33-ST2-CXCL3−CXCR2 axis in PDAC metastasis was evaluated in three clinically relevant mouse PDAC models.
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Eradication of tumor growth by delivering novel photothermal selenium-coated tellurium nanoheterojunctions
Shiyou Chen,Shiyou Chen,Shiyou Chen,Chenyang Xing,Dazhou Huang,Chuanhong Zhou,Bo Ding,Ziheng Guo,Zhengchun Peng,Dou Wang,Xi Zhu,Shuzhen Liu,Zhen Cai,Jieyu Wu,Jiaqi Zhao,Zongze Wu,Yuhua Zhang,Chaoying Wei,Yan Qiaoting,Hongzhong Wang,Dianyuan Fan,Liping Liu,Han Zhang,Yihai Cao +23 more
TL;DR: A stable, highly uniform in size, and nontoxic nanomaterials made of tellurium-selenium (TeSe)–based lateral heterojunction are developed that nearly completely eradicated lung cancer and hepatocellular carcinoma in preclinical models.
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Prodrug-Loaded Zirconium Carbide Nanosheets as a Novel Biophotonic Nanoplatform for Effective Treatment of Cancer.
Quan Liu,Zhongjian Xie,Zhongjian Xie,Meng Qiu,Meng Qiu,Inseob Shim,Yunlong Yang,Yunlong Yang,Sisi Xie,Qinhe Yang,Dou Wang,Shiyou Chen,Taojian Fan,Bo Ding,Ziheng Guo,Dickson Adah,Dickson Adah,Xinhuang Yao,Yuhua Zhang,Hong Wu,Zongze Wu,Chaoying Wei,Hongzhong Wang,Hyeong Seok Kim,Qingshuang Zou,Yan Qiaoting,Zhen Cai,Jong Seung Kim,Liping Liu,Han Zhang,Yihai Cao +30 more
TL;DR: In vitro and in vivo experimental evidence shows the potent anticancer effects of the integrated ZrC@prodrug biophotonic nanoplatform by specifically targeting malignant cells, chemotherapy/hyperthermia‐induced tumor inflammation, and angiogenesis.
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Off-tumor Targets Compromise Antiangiogenic Drug Sensitivity by Inducing Kidney Erythropoietin Production
Masaki Nakamura,Yin Zhang,Yin Zhang,Yunlong Yang,Ceylan Sonmez,Wenyi Zheng,Guichun Huang,Takahiro Seki,Hideki Iwamoto,Bo Ding,Linlin Yin,Theodoros Foukakis,Thomas Hatschek,Xuri Li,Kayoko Hosaka,Jiaping Li,Guohua Yu,Xinsheng Wang,Yizhi Liu,Yihai Cao +19 more
TL;DR: It is shown that in mouse tumor models and in human cancer patients, the anti-VEGF drug-induced kidney hypoxia augments circulating levels of erythropoietin (EPO) and uncover a mechanism of desensitizing antiangiogenic and anticancer effects by kidney-produced EPO.
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