Björn Andersson
Sahlgrenska University Hospital
41 Papers
202 Citations
Björn Andersson is an academic researcher from Sahlgrenska University Hospital. The author has contributed to research in topics: Heart failure & Stroke. The author has an hindex of 16, co-authored 41 publications.
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Papers
Mapping of a functional autoimmune epitope on the beta 1-adrenergic receptor in patients with idiopathic dilated cardiomyopathy.
Yvonne Magnusson,Stefano Marullo,S Hoyer,Finn Waagstein,Björn Andersson,A Vahlne,Jean-Gérard Guillet,Arthur Donny Strosberg,A Hjalmarson,Johan Hoebeke +9 more
TL;DR: It is concluded that a subgroup of patients with idiopathic dilated cardiomyopathy have in their sera autoantibodies specifically directed against the second extracellular loop of the beta 1-adrenergic receptor.
The activity of the hypothalamic-pituitary-adrenal axis and the sympathetic nervous system in relation to waist/hip circumference ratio in men.
Thomas Ljung,Göran Holm,Peter Friberg,Björn Andersson,Bengt-Åke Bengtsson,Johan Svensson,Mary F. Dallman,Bruce S. McEwen,Per Björntorp +8 more
TL;DR: A mild dysregulation of the HPA axis is suggested, occurring with elevated WHR independent of the BMI, and a central activation of the sympathetic nervous system is indicated, such as in the early phases of hypertension, correlating with insulin resistance.
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Neuronal and Glia-Related Biomarkers in Cerebrospinal Fluid of Patients with Acute Ischemic Stroke
Clara Hjalmarsson,Maria Bjerke,Björn Andersson,Kaj Blennow,Henrik Zetterberg,N. David Åberg,Bob Olsson,Carl Eckerström,Lena Bokemark,Anders Wallin +9 more
- 19 May 2014
TL;DR: Several CSF biomarkers increase in AIS, and they correlate to clinical stroke severity, however, only NFL was found to be a marker of degree of WML.
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Effects of pacing-induced myocardial stress and spinal cord stimulation on whole body and cardiac norepinephrine spillover
H. Norrsell,T. Eliasson,C. Mannheimer,L.-E. Augustinsson,Claes-Håkan Bergh,Björn Andersson,Finn Waagstein,Peter Friberg +7 more
TL;DR: The results of this study indicate that the anti-ischaemic effect of spinal cord stimulation is not due to reduced cardiac sympathetic activity, however, spinal Cord stimulation decreases overall sympathetic activity which may benefit the heart, possibly by reducing oxygen demand.
Different responses to dobutamine in the presence of carvedilol or metoprolol in patients with chronic heart failure
TL;DR: Patients with congestive heart failure on a non-selective β adrenoreceptor blocker or β1 selective blocker responded differently to the inotropic drug dobutamine: the β1 blockade caused by metoprolol could be counteracted by do butamine, whereas with carvedilol a low dose of dobutamines increased cardiac output, and a higher dose ofDobutamine caused a pressor effect.
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