Birgit Lange
10 Papers
9 Citations
Birgit Lange is an academic researcher. The author has contributed to research in topics: Systemic acquired resistance & Arabidopsis. The author has an hindex of 6, co-authored 9 publications.
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Papers
Systemic acquired resistance networks amplify airborne defense cues.
Marion Wenig,Andrea Ghirardo,Jennifer Sales,Elisabeth Pabst,Heiko H. Breitenbach,Felix Antritter,Baris Weber,Birgit Lange,Miriam Lenk,Robin K. Cameron,Joerg-Peter Schnitzler,A. Corina Vlot +11 more
TL;DR: It is shown that monoterpene-associated responses are propagated in feed-forward loops involving the systemic acquired resistance (SAR) signaling components pipecolic acid, glycerol-3-phosphate, and LEGUME LECTIN-LIKE PROTEIN1 (LLP1).
UGT76B1, a promiscuous hub of small molecule-based immune signaling, glucosylates N-hydroxypipecolic acid, and balances plant immunity.
Sibylle Bauer,Dereje W. Mekonnen,Michael Hartmann,Ipek Yildiz,Robert Janowski,Birgit Lange,Birgit Geist,Jürgen Zeier,Anton R. Schäffner +8 more
TL;DR: The authors showed that UGT76b1 also glucosylates N-hydroxypipecolic acid (NHP), which is synthesized by FLAVIN-DEPENDENT MONOOXYGENASE 1 (FMO1) and activates systemic acquired resistance (SAR).
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Metabolomic adjustments in the orchid mycorrhizal fungus Tulasnella calospora during symbiosis with Serapias vomeracea.
Andrea Ghirardo,Valeria Fochi,Valeria Fochi,Birgit Lange,Michael Witting,Jörg-Peter Schnitzler,Silvia Perotto,Silvia Perotto,Raffaella Balestrini +8 more
TL;DR: Novel insights are provided on the mechanisms underlying the orchid mycorrhizal association and intriguing questions are opened on the role of fungal lipids in this symbiosis.
UGT76B1, a promiscuous hub of small molecule-based immune signaling, glucosylates N-hydroxypipecolic acid and controls basal pathogen defense
Sibylle Bauer,Dereje W. Mekonnen,Michael Hartmann,Robert Janowski,Birgit Lange,Birgit Geist,Jürgen Zeier,Anton R. Schäffner +7 more
TL;DR: It is shown that UGT76B1 also glucosylates N-hydroxypipecolic acid (NHP), which is synthesized by FLAVIN-DEPENDENT MONOOXYGENASE 1 (FMO1) and activates systemic acquired resistance (SAR), and that ILA promotes and SA finally executes the NHP-triggered immunity via the glucOSyltransferase UGT 76B1 as the common metabolic hub.
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Mutant Muddle: Some Arabidopsis eds5 Mutant Lines Have a Previously Unnoticed Second-Site Mutation in FAH1.
Sravani Ram Veeragoni,Birgit Lange,Mario Serrano,Christiane Nawrath,Sibylle Bauer,Anton R. Schäffner,Hans Thordal-Christensen,Jörg Durner,Frank Gaupels +8 more
TL;DR: Some of the salicylic acid-deficient Arabidopsis eds5 mutants have an unnoticed fah1-2 background mutation, which could cause salicyric acid- and EDS5-independent mutant phenotypes.
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