Bin Que
Capital Medical University
45 Papers
33 Citations
Bin Que is an academic researcher from Capital Medical University. The author has contributed to research in topics: Medicine & Myocardial infarction. The author has an hindex of 8, co-authored 19 publications.
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Papers
Interleukin-12p35 Knock Out Aggravates Doxorubicin-Induced Cardiac Injury and Dysfunction by Aggravating the Inflammatory Response, Oxidative Stress, Apoptosis and Autophagy in Mice.
Jing Ye,Ying Huang,Bin Que,Chao Chang,Wenjing Liu,Haiying Hu,Ling Liu,Ying Shi,Yuan Wang,Menglong Wang,Tao Zeng,Wang Zhen,Yao Xu,Lei Shi,Jianfang Liu,Huimin Jiang,Di Ye,Yingzhong Lin,Jun Wan,Qingwei Ji +19 more
TL;DR: IL-12p35 KO aggravated DOX-induced cardiac injury by amplifying the levels of inflammation, oxidative stress, apoptosis and autophagy.
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Circulating Th1, Th2, and Th17 Levels in Hypertensive Patients
Qingwei Ji,Guojie Cheng,Ning Ma,Ying Huang,Yingzhong Lin,Qi Zhou,Bin Que,Jian-Zeng Dong,Yujie Zhou,Shaoping Nie +9 more
TL;DR: The changes in Th1, Th2, and Th17 activity are associated with the onset of the nondipper type and carotid atherosclerotic plaque in hypertensive patients.
Interleukin-12p35 knockout promotes macrophage differentiation, aggravates vascular dysfunction, and elevates blood pressure in angiotensin II-infused mice.
Jing Ye,Jing Ye,Bin Que,Ying Huang,Yingzhong Lin,Jiangbin Chen,Ling Liu,Ying Shi,Yuan Wang,Menglong Wang,Tao Zeng,Zhen Wang,Haiying Hu,Yao Xu,Lei Shi,Di Ye,Jianfang Liu,Huimin Jiang,Jun Wan,Qingwei Ji +19 more
TL;DR: IL-12p35 KO amplifies the inflammatory response and promotes blood pressure elevation in Ang II-treated mice, and IL-12, but not IL-35, plays a protective role in theAng II-induced hypertension model, suggesting IL- 12 may be a novel therapeutic agent for the prevention and treatment of clinical hypertension.
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Association of obstructive sleep apnoea with cardiovascular events in women and men with acute coronary syndrome
Xiaobo Wang,Jing-Yao Fan,Ruifeng Guo,Wen Hao,Wei Gong,Yang Yang,Wen-Bin Zheng,Hui Ai,Bin Que,Dan-Hong Hu,Changsheng Ma,Xin-Liang Ma,Virend K. Somers,Shaoping Nie +13 more
TL;DR: In hospitalized ACS patients, OSA was associated with increased risk of subsequent events, particularly among women, and female patients with ACS should be neglected for OSA screening and dedicated intervention studies focusing on women with ACS and comorbid OSA should be prioritized.
IL-37 inhibits the maturation of dendritic cells through the IL-1R8-TLR4-NF-κB pathway.
Tianxiao Liu,Jing Liu,Yingzhong Lin,Bin Que,Chao Chang,Jianwei Zhang,Zhishan Liang,Xiaonan Gao,Shangwei Liu,Ling Liu,Dazhu Li,Qingwei Ji +11 more
TL;DR: This study indicated that IL-37 inhibited the maturation of DCs via the IL-1R8-TLR4-NF-κB pathway and attenuated atherosclerosis in ApoE-/- mice.
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