14 Papers
16 Citations
Bin Dai is an academic researcher from Fourth Military Medical University. The author has contributed to research in topics: Medicine & Cancer. The author has an hindex of 10, co-authored 11 publications.
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Papers
Long noncoding RNA SLC2A1-AS1 regulates aerobic glycolysis and progression in hepatocellular carcinoma via inhibiting the STAT3/FOXM1/GLUT1 pathway.
Runze Shang,Miao Wang,Bin Dai,Jianbing Du,Jianlin Wang,Zekun Liu,Shibin Qu,Xisheng Yang,Jingjing Liu,Congcong Xia,Lin Wang,Desheng Wang,Yu Li +12 more
TL;DR: It is reported that lncRNA SLC2A1‐AS1 is frequently downregulated in HCC samples, as shown by quantitative real‐time polymerase chain reaction analysis and significantly suppressed glycolysis of HCC.
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MicroRNA-150 suppresses cell proliferation and metastasis in hepatocellular carcinoma by inhibiting the GAB1-ERK axis
Wei Sun,Zhuochao Zhang,Jianlin Wang,Runze Shang,Liang Zhou,Xing Wang,Juanli Duan,Bai Ruan,Yuan Gao,Bin Dai,Shibin Qu,Wei Liu,Rui Ding,Lin Wang,Desheng Wang,Kefeng Dou +15 more
TL;DR: It is found that miR-150 was significantly downregulated in hepatocellular carcinoma (HCC) tissues compared to adjacent noncancerous tissues and that the identified miR -150-GAB1-ERK axis is a potential therapeutic target for HCC.
A review of pig liver xenotransplantation: Current problems and recent progress
Xuan Zhang,Xiao Li,Zhaoxu Yang,Kai-shan Tao,Quancheng Wang,Bin Dai,Shibin Qu,Wei Peng,Hong Zhang,David K. C. Cooper,Ke-Feng Dou +10 more
TL;DR: Clinical trials of pig liver xenotransplantation should initially be considered as a bridge to allotransplantations, with the aim of identifying the ideal transgene combination for pig liver Xenotrans transplantation.
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RRAD inhibits aerobic glycolysis, invasion, and migration and is associated with poor prognosis in hepatocellular carcinoma.
Runze Shang,Jianlin Wang,Wei Sun,Bin Dai,Bai Ruan,Zhuochao Zhang,Xisheng Yang,Yuan Gao,Shibin Qu,Xing Lv,Kaishan Tao,Lin Wang,Kefeng Dou,Desheng Wang +13 more
TL;DR: It is revealed that RRAD expression was decreased in HCC tumor tissues and predicted poor clinical outcome for HCC patients and played an important role in regulating aerobic glycolysis and cell invasion and metastasis and may represent potential targets for improving the treatment of HCC.
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Tg737 regulates epithelial-mesenchymal transition and cancer stem cell properties via a negative feedback circuit between Snail and HNF4α during liver stem cell malignant transformation.
Qike Huang,Meng Pu,Ge Zhao,Bin Dai,Zhenyuan Bian,Haili Tang,Chong Chen,Wei Liu,Xuan Qu,Liangliang Shen,Kaishan Tao +10 more
TL;DR: Results showed that Tg737 regulates a Wnt/β-catenin/Snail-HNF4α negative feedback circuit, thereby blocking EMT and the malignant transformation of liver stem cells to liver cancer stem cells.
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