Béatrice Benoit
Université Paris-Saclay
6 Papers
10 Citations
Béatrice Benoit is an academic researcher from Université Paris-Saclay. The author has contributed to research in topics: Septin & Microtubule. The author has an hindex of 2, co-authored 6 publications. Previous affiliations of Béatrice Benoit include French Institute of Health and Medical Research.
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Papers
Stress-induced phosphorylation of CLIP-170 by JNK promotes microtubule rescue.
Hélène Henrie,Dalal Bakhos-Douaihy,Isabelle Cantaloube,Antoine Pilon,Antoine Pilon,Maya Talantikite,Virginie Stoppin-Mellet,Anita Baillet,Christian Poüs,Christian Poüs,Béatrice Benoit +10 more
TL;DR: Upon stress, JNK stimulates microtubule dynamics in human epithelial cell lines and phosphorylates the rescue factor CLIP-170, increasing its retention frequency on the lattice behind comets, at locations that correspond to potential future rescue sites.
MAPping the Wnt pathway to hepatocellular carcinoma recurrence.
Béatrice Benoit,Christian Poüs +1 more
TL;DR: Early recurrence is a frequent cause of poor survival after hepatocellular carcinoma (HCC) resection and efforts to link HCC with the dysfunction of signalling pathways identified several alterations, especially in the Wnt/β-catenin pathway.
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Microtubule reorientation in the blue spotlight: Cutting and CLASPing at dynamic hot spots.
Béatrice Benoit,Christian Poüs +1 more
TL;DR: Benoit and Poüs highlight new work from Lindeboom et al. revealing how CLASP reorients microtubule networks in plants growing toward light.
Septin filament coalignment with microtubules depends on SEPT9_i1 and tubulin polyglutamylation, and is an early feature of acquired cell resistance to paclitaxel.
Benjamin Targa,Laurence Klipfel,Isabelle Cantaloube,Joëlle Salameh,Béatrice Benoit,Christian Poüs,Anita Baillet +6 more
TL;DR: Coupling septins (including SEPT9_i1) overexpression together with long-chain tubulin polyglutamylation induce significant paclitaxel resistance in several naive (taxane-sensitive) cell lines and accordingly stimulate the binding of CLIP-170 and MCAK to microtubules.
Cytoskeleton and Associated Proteins: Pleiotropic JNK Substrates and Regulators.
TL;DR: In this paper, the complex relationship between the stress-induced c-Jun N-terminal kinases (JNKs) and the four main cytoskeleton elements, which are actin filaments, microtubules, intermediate filaments and septins, is discussed.