Beate Geist
Charité
7 Papers
69 Citations
Beate Geist is an academic researcher from Charité. The author has contributed to research in topics: Gene & Internal medicine. The author has an hindex of 5, co-authored 7 publications. Previous affiliations of Beate Geist include University of Mainz.
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Papers
Kidney-Specific Inactivation of the Megalin Gene Impairs Trafficking of Renal Inorganic Sodium Phosphate Cotransporter (NaPi-IIa)
Sebastian Bachmann,Uwe Schlichting,Beate Geist,Kerim Mutig,Thomas Petsch,Desa Bacic,Carsten A. Wagner,Brigitte Kaissling,Jürg Biber,Heini Murer,Thomas E. Willnow +10 more
TL;DR: Investigation was the role of megalin in the regulation of NaPi-IIa in steady state and during inactivation, and the capacity of the proximal tubule cell to react on PTH-driven inactivation of Na Pi- IIa by endocytosis and intracellular translocation require the presence of meGalin.
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Role of lipid rafts in membrane delivery of renal epithelial Na+-K+-ATPase, thick ascending limb.
Pia Welker,Beate Geist,Jan-Henning Frühauf,Michele Salanova,David A. Groneberg,Eberhard Krause,Sebastian Bachmann +6 more
TL;DR: Results from isolated lipid rafts from rat kidney by sucrose gradient fractionation after detergent treatment may have functional implications regarding the turnover, trafficking, and regulated surface expression of NKA as the major basolateral ion transporter of TAL.
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Plasticity-related gene 3 promotes neurite shaft protrusion
Tanja Velmans,Arne Battefeld,Arne Battefeld,Beate Geist,Anna Soriguera Farrés,Anna Soriguera Farrés,Ulf Strauss,Anja U. Bräuer +7 more
TL;DR: It is demonstrated here that PRG3 induces filopodia formation in HEK293 cells depending on its N-glycosylation status and contributes to regulating filo-temporal formation in immature neurons.
PRG-1 transcriptional regulation independent from Nex1/Math2-mediated activation.
TL;DR: It is found that PRG-1 expression is under the control of a TATA-less promoter with multiple transcription start sites, mediating ≈40-fold enhancement of transcription in cultured primary neurons compared to controls, and which induced reporter expression in slice cultures in neurons.
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Specific properties of sodium currents in multipotent striatal progenitor cells.
Ulrike Wasner,Beate Geist,Arne Battefeld,Peter Bauer,Jana Müller,Arndt Rolfs,Ulf Strauss,Ulf Strauss +7 more
TL;DR: It is concluded that multipotent neural progenitor cells express Na+ channels in their membrane irrespective of their fate but these channels have little effect due to their subunit composition, which is regulated by alternative splicing.
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