Atsushi Kawakami
Nagasaki University
717 Papers
3.8K Citations
Atsushi Kawakami is an academic researcher from Nagasaki University. The author has contributed to research in topics: Medicine & Rheumatoid arthritis. The author has an hindex of 50, co-authored 663 publications. Previous affiliations of Atsushi Kawakami include RMIT University.
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Papers
Pilot Trial on the Effect of 5-Aminolevulinic Acid on Glucose Tolerance in Patients with Maternally Inherited Diabetes and Deafness
TL;DR: In this article , the amino acid 5-aminolevulinic acid (5-ALA) was combined with sodium ferrous citrate (SFC) to increase ATP production in mitochondria.
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Subacute thyroiditis in a patient with psoriatic arthritis switched from secukinumab to adalimumab: a case report and literature review
Jun Nakagawa,K. Fujikawa,Midori Akagi,Keita Nakaji,Junichi Yasui,Yumi Hanatani,Toshihide Hara,Akinari Mizokami,Atsushi Kawakami +8 more
- 02 Jan 2021
TL;DR: Following the administration of ixekizumab, an IL-17A inhibitor, her PsA improved without SAT relapse, and SAT mechanism associated with TNF inhibitors remains unknown, but cytokine imbalance may be involved.
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CD4+ T-cell-mediated cytotoxicity against staphylococcal enterotoxin B-pulsed synovial cells.
Atsushi Kawakami,Naoki Matsuoka,Masahiko Tsuboi,Satoshi Urayama,Tomoki Nakashima,Yojiro Kawabe,Takehiko Koji,Takahiko Aoyagi,K Maeda,K Eguchi +9 more
TL;DR: The results suggest that CD4+ T’cells are activated by synovial cells by an SEB‐dependent manner and express FasL, inducing Fas‐mediated apoptosis of the latter cells, which may regulate the overgrowth of synovia cells in RA synovium.
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Certolizumab pegol was effective for treating residual synovitis after total knee arthroplasty in a patient with rheumatoid arthritis: therapeutic monitoring by ultrasound.
TL;DR: A 59-year-old female who developed rheumatoid arthritis in 2007 is presented, who was treated with methotrexate (MTX) after the operation, and achieved clinical remission.
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Clinical Characteristics of Patients With IgG4-Related Disease Complicated by Hypocomplementemia
Yuya Fujita,Shoichi Fukui,Masataka Umeda,Sosuke Tsuji,Naoki Iwamoto,Yoshikazu Nakashima,Yoshiro Horai,Takahisa Suzuki,Akitomo Okada,Toshiyuki Aramaki,Yukitaka Ueki,Akinari Mizokami,Tomoki Origuchi,Hiroshi Watanabe,Kiyoshi Migita,Atsushi Kawakami +15 more
TL;DR: Patients with IgG4-RD with hypocomplementemia have a more active clinical phenotype, suggesting contributions of the complement system in the pathophysiology of IgG 4-RD.
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