Asiya Batool
University of Kashmir
17 Papers
25 Citations
Asiya Batool is an academic researcher from University of Kashmir. The author has contributed to research in topics: EIF4E & mTORC1. The author has an hindex of 7, co-authored 14 publications. Previous affiliations of Asiya Batool include Council of Scientific and Industrial Research.
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Papers
TGF-β signaling: A recap of SMAD-independent and SMAD-dependent pathways.
Sabreena Aashaq,Asiya Batool,Shabir Ahmad Mir,Mushtaq A. Beigh,Khurshid Iqbal Andrabi,Zaffar Amin Shah +5 more
TL;DR: In this article, the authors make an attempt to summarize the TGF-β-mediated SMAD-dependent and SMADindependent pathways and emphasize the functions of TGFβ as a metastatic promoter and tumor suppressor during the later and initial phases of tumor progression respectively.
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TAK1 mediates convergence of cellular signals for death and survival
TL;DR: This review highlights how TAK1 orchestrates regulation of energy homeostasis via AMPK and its emerging role in influencing mTORC1 pathway to regulate death or survival in tandem.
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Three months of COVID-19: A systematic review and meta-analysis.
TL;DR: Some recently proposed conceptual models that estimate the spread of this disease based on the basic reproductive number for better prevention and control procedures are shed light on how the virus has endangered the global economy.
54
Eukaryotic initiation factor 4E (eIF4E): A recap of the cap-binding protein
TL;DR: This review highlights the latest studies that have envisioned to target these pathways and ultimately the translational machinery for therapeutic intervention and brings forward the prospect of eIF4E to act as a converging juncture for signaling pathways like mTOR/PI3K and Mnk/MAPK to promote tumorigenesis.
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Reappraisal to the study of 4E-BP1 as an mTOR substrate - A normative critique.
TL;DR: This review provides new insights into the molecular network of signalling pathways highlighting the recent explosion of studies in respect to the deviant behaviour of 4E-BP1 towards mTORC1, and brings forward comprehensive studies delineating the redundancy of 4e-BP isoforms in regulating translational control.
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