Aric Huang
McMaster University
13 Papers
4 Citations
Aric Huang is an academic researcher from McMaster University. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 6, co-authored 7 publications.
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Papers
Untargeted Metabolomics in the Discovery of Novel Biomarkers and Therapeutic Targets for Atherosclerotic Cardiovascular Diseases.
TL;DR: There is a need to establish sensitive and non-invasive CV biomarkers, and novel therapeutic targets for the prevention and treatment of CVDs, and there remains a need for future advancement in metabolomic technologies.
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Deletion of Myeloid GSK3α Attenuates Atherosclerosis and Promotes an M2 Macrophage Phenotype
Cameron S. McAlpine,Aric Huang,Abby Emdin,Nicole S. Banko,Daniel R. Beriault,Yuanyuan Shi,Geoff H. Werstuck +6 more
TL;DR: The findings suggest that deletion of myeloid GSK3&agr; attenuates the progression of atherosclerosis by promoting an M2 macrophage phenotype.
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Molecular probes of spike ectodomain and its subdomains for SARS-CoV-2 variants, Alpha through Omicron
I-Ting Teng,Alexandra Nazzari,Misook Choe,Tracy L. McGaha Liu,Matheus Oliveira de Souza,Yuliya Petrova,Yaroslav Tsybovsky,Shuishu Wang,Bao Shan Zhang,Mykhaylo V. Artamonov,Bharat Madan,Aric Huang,Sheila N. Lopez Acevedo,Xiaoli Pan,Tracy J. Ruckwardt,Brandon J. DeKosky,John R. Mascola,John Misasi,Nancy J. Sullivan,Tongqing Zhou,Peter D. Kwong +20 more
TL;DR: A matrix of SARS-CoV-2 variant molecular probes are described that allow for assessment of immune responses, identification of serum antibody specificity, and isolation and characterization of neutralizing antibodies.
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The Role of Endoplasmic Reticulum Stress-Glycogen Synthase Kinase-3 Signaling in Atherogenesis
TL;DR: The knowledge of the potential role of ER stress-GSK3 signaling in the development and progression of atherosclerosis as well as the possible therapeutic implications of this pathway are summarized.
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•Journal Article
Abstract 19: Deletion of Myeloid GSK3α Attenuates Atherosclerosis and Promotes an M2 Macrophage Phenotype
Cameron S. McAlpine,Aric Huang,Abby Emdin,Nicole S. Banko,Daniel R. Beriault,Yuanyuan Shi,Geoff H. Werstuck +6 more
TL;DR: The findings suggest that deletion of myeloid GSK3α attenuates the progression of atherosclerosis by regulating STAT activation and promoting an M2 macrophage phenotype.
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