Antti Lauerma
University of Helsinki
134 Papers
751 Citations
Antti Lauerma is an academic researcher from University of Helsinki. The author has contributed to research in topics: Medicine & Atopic dermatitis. The author has an hindex of 36, co-authored 122 publications. Previous affiliations of Antti Lauerma include Helsinki University Central Hospital & University of Osnabrück.
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Papers
IL-31: A new link between T cells and pruritus in atopic skin inflammation
Enikö Sonkoly,Anja Müller,Antti Lauerma,Andor Pivarcsi,Hortensia Soto,Lajos Kemény,Harri Alenius,Marie-Caroline Dieu-Nosjean,Stephan Meller,Juliane Rieker,Martin Steinhoff,Thomas K. Hoffmann,Thomas Ruzicka,Albert Zlotnik,Bernhard Homey +14 more
TL;DR: The findings provide a new link among staphylococcal colonization, subsequent T-cell recruitment/activation, and pruritus induction in patients with atopic dermatitis, and show that IL-31 may represent a novel target for antipruritic drug development.
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CCL27-CCR10 interactions regulate T cell-mediated skin inflammation.
Bernhard Homey,Harri Alenius,Anja Müller,Hortensia Soto,Edward P. Bowman,Wei Yuan,Leslie McEvoy,Antti Lauerma,Till Assmann,Erich Bünemann,Maili Lehto,Henrik Wolff,David C. Yen,Heather Marxhausen,Wayne To,Jonathon D. Sedgwick,Thomas Ruzicka,Percy Lehmann,Albert Zlotnik +18 more
TL;DR: It is reported that most skin-infiltrating lymphocytes in patients suffering from psoriasis, atopic or allergic-contact dermatitis express CCR10, indicating that CCL27–CCR10 interactions have a pivotal role in T cell–mediated skin inflammation.
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The international EAACI/GA²LEN/EuroGuiDerm/APAAACI guideline for the definition, classification, diagnosis, and management of urticaria.
Torsten Zuberbier,Amir Hamzah Abdul Latiff,Mohamed Abuzakouk,S. Aquilina,Riccardo Asero,Diane Baker,Barbara Ballmer-Weber,Barbara Ballmer-Weber,Christine Bangert,Moshe Ben-Shoshan,Jonathan A. Bernstein,Carsten Bindslev-Jensen,Knut Brockow,Zenon Brzoza,Herberto José Chong Neto,Martin K. Church,Martin K. Church,Paulo Ricardo Criado,I V Danilycheva,Corinna Dressler,Luis Felipe Ensina,Luz Fonacier,Matthew Gaskins,Krisztián Gáspár,Aslı Gelincik,A. Giménez-Arnau,Kiran Godse,Margarida Gonçalo,Clive Grattan,Martine Grosber,Eckard Hamelmann,Jacques Hébert,Michihiro Hide,Allen P. Kaplan,Alexander Kapp,Aharon Kessel,Emek Kocatürk,Kanokvalai Kulthanan,Désirée Larenas-Linnemann,Antti Lauerma,Tabi A. Leslie,Markus Magerl,Markus Magerl,Michael Makris,R. Y. Meshkova,Martin Metz,Martin Metz,Daniel Micallef,Charlotte G. Mortz,Alexander Nast,Hanneke Oude-Elberink,Ruby Pawankar,Paolo D. Pigatto,Hector Ratti Sisa,María Isabel Rojo Gutiérrez,Sarbjit S. Saini,Peter Schmid-Grendelmeier,Bulent Enis Sekerel,Frank Siebenhaar,Frank Siebenhaar,Hanna Siiskonen,Angèle Soria,Petra Staubach-Renz,Luca Stingeni,Gordon Sussman,Andrea Szegedi,Simon Francis Thomsen,Zahava Vadasz,Christian Vestergaard,Bettina Wedi,Zuotao Zhao,Marcus Maurer,Marcus Maurer +72 more
TL;DR: This guideline was acknowledged and accepted by the European Union of Medical Specialists (UEMS) on 3 December 2020 as mentioned in this paper, with the participation of 64 delegates of 50 national and international societies and from 31 countries.
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IL-33 and ST2 in Atopic Dermatitis: Expression Profiles and Modulation by Triggering Factors
Terhi Savinko,Sampsa Matikainen,Ulpu Saarialho-Kere,Maili Lehto,Guoying Wang,Sari Lehtimäki,Piia Karisola,Timo Reunala,Henrik Wolff,Antti Lauerma,Antti Lauerma,Antti Lauerma,Harri Alenius +12 more
TL;DR: Increased expression of IL-33 and ST2 in AD skin after allergen or staphylococcal enterotoxin B (SEB) exposure is found as well as in the skin of 22-week-old filaggrin-deficient mice, suggesting an important role for IL- 33-ST2 interaction in AD and highlighting the fact that bacterial and viral infections may increase the production ofIL-33.
366
Plasmacytoid dendritic cells sense skin injury and promote wound healing through type I interferons.
Josh Gregorio,Josh Gregorio,Stephan Meller,Curdin Conrad,Anna Di Nardo,Bernhard Homey,Antti Lauerma,Naoko Arai,Richard L. Gallo,John DiGiovanni,Michel Gilliet,Michel Gilliet +11 more
TL;DR: Cutaneous injury in mice drives transient TLR7- and TLR9-mediated production of type I interferon by plasmacytoid dendritic cells, which is required for re-epithelialization of the skin.
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