Ann McCormack
National Institutes of Health
32 Papers
266 Citations
Ann McCormack is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Transplantation & Endothelial stem cell. The author has an hindex of 14, co-authored 30 publications. Previous affiliations of Ann McCormack include Imperial College London & Harefield Hospital.
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Papers
A comparison of primary endothelial cells and endothelial cell lines for studies of immune interactions.
TL;DR: Significant differences were found in the ability to respond to cytokines between HUVEC and the cell lines, the greatest differences being induction of VCAM-1 and E-selectin in response to TNF-alpha and induction of MHC class II antigens in Response to IFN-gamma.
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MHC class II expression on human heart microvascular endothelial cells: exquisite sensitivity to interferon-gamma and natural killer cells.
TL;DR: Compared the sensitivity of human heart MVEC, human umbilical vein endothelial cells (HUVEC), and adult large vessel EC to interferon (IFN)-gamma and natural killer (NK) cell-mediated induction of MHC class II antigens, results suggest that basal expression in vivo is maintained by circulating factors.
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Protective effect of phosphorylated Hsp27 in coronary arteries through actin stabilization
TL;DR: The results are consistent with the hypothesis that phospho-Hsp27 protects against vascular disease possibly by stabilizing the actin cytoskeleton within endothelial and/or smooth muscle cells.
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Lack of effect of MICA antibodies on graft survival following heart transplantation.
John D. Smith,V. M. Brunner,S. Jigjidsuren,I. M. Hamour,Ann McCormack,N.R. Banner,Marlene L. Rose +6 more
TL;DR: This study suggests that MICA Abs do not adversely affect the outcome of cardiac transplantation.
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The effects of monocytes on the transendothelial migration of T lymphocytes
TL;DR: Investigation of the role of monocytes on T‐cell migration and induction of endothelial adhesion molecules concluded that monocytes, via cell surface or released cytokines play an essential role in allowing large‐scale recruitment of T cells to inflammatory sites in vivo.
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