Andrew H. Miller
Emory University
336 Papers
1.8K Citations
Andrew H. Miller is an academic researcher from Emory University. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 90, co-authored 307 publications. Previous affiliations of Andrew H. Miller include Icahn School of Medicine at Mount Sinai & University of Texas at Arlington.
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Papers
Depression and Inflammation in Patients With Lung Cancer: A Comparative Analysis of Acute Phase Reactant Inflammatory Markers.
TL;DR: Positive and negative APRs may form a distinct biologic signature to help identify patients with inflammatory depression in the lung cancer setting and albumin is associated with depression but not to a greater extent than CRP.
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Pilot study of combined aerobic and resistance exercise on fatigue for patients with head and neck cancer: Inflammatory and epigenetic changes
Canhua Xiao,Jonathan J. Beitler,Kristin Higgins,Cynthia E Chico,Janice S. Withycombe,Ying Zhu,Hongyu Zhao,I-Hsin Lin,Fangyong Li,Sangchoon Jeon,Melinda L. Irwin,Deborah Watkins Bruner,Andrew H. Miller,Rebecca A. Gary +13 more
TL;DR: It is suggested that exercise may improve physical performance and reduce fatigue, which could be further linked to decreased inflammation, during active radiotherapy for HNC patients.
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Lymphocytic responses and the gradual hippocampal neuron loss following infection with lymphocytic choriomeningitis virus (LCMV).
TL;DR: The data suggest that T lymphocytes control glial infection and mediate degeneration in the cerebellum but not the hippocampus, which is known to cause a bi-phasic neurodegeneration characterized by acute T lymphocyte-mediated cerebellar damage.
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Influence of desmethylimipramine on natural killer cell activity.
TL;DR: In vitro results demonstrate that DMI reversibly inhibits NK activity at serum concentrations that are not uncommonly found in depressed patients receiving this medication.
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Elucidating the consequences of chronic stress on immune regulation and behavior in rheumatoid arthritis.
TL;DR: Chronic psychological stress is associated with increased in vitro stimulation of IL-6 and decreased sensitivity of in vitro stimulated IL- 6 production to inhibition by glucocorticoids, thereby potentially limiting the beneficial antiinflammatory effects of endogenous, and possibly exogenous, glucocORTicoids.
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