Andrea Szegedi
University of Debrecen
163 Papers
871 Citations
Andrea Szegedi is an academic researcher from University of Debrecen. The author has contributed to research in topics: Medicine & Atopic dermatitis. The author has an hindex of 28, co-authored 144 publications. Previous affiliations of Andrea Szegedi include Hungarian Academy of Sciences.
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Papers
Extended diagnostic value of autologous serum skin test and basophil CD63 expression assay in chronic urticaria.
Beatrix Irinyi,Edit Gyimesi,Edina Garaczi,Zsuzsanna Bata,Lajos Kemény,Margit Zeher,Éva Remenyik,Andrea Szegedi +7 more
TL;DR: Readers should note that articles published below have been fully refereed, but have not been through the copy-editing and proof correction process.
Dermatology Life Quality Index (DLQI) score bands are applicable to DLQI-Relevant (DLQI-R) scoring.
Fanni Rencz,Fanni Rencz,L.H. Gergely,Norbert Wikonkál,Krisztián Gáspár,Márta Péntek,László Gulácsi,Béla Tamási,A.K. Poór,Ágnes Kinyó,Gábor Bali,Bernadett Hidvégi,Miklós Sárdy,K. Hajdu,Andrea Szegedi,Éva Remenyik,Zsuzsanna Bata-Csörgő,Péter Holló,Petra Baji,Valentin Brodszky +19 more
TL;DR: DLZI-Relevant (DLQI-R) is a recently developed scoring modification of the Dermatology Life Quality Index that improved convergent validity, discriminatory power and responsiveness of the questionnaire.
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Isotretinoin is indirectly effective in sebocytes.
Dóra Kovács,Katalin Hegyi,Andrea Szegedi,Dávid Deák,Sz. Póliska,Ralph Rühl,Christos C. Zouboulis,Dániel Törőcsik +7 more
TL;DR: In a recently published research letter Burney et al. demonstrated that isotretinoin (13-cis-retinoic acid, 13CRA) induced acnegenic changes in SEB-1 cultured human sebocytes1, but suggested that the observed changes may be influenced by the Simian virus (SV)-40 immortalization of the used cell line.
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Heme Oxygenase and the Skin in Health and Disease
TL;DR: The role of HO in the skin in physiological conditions as well as in relevant dermatological diseases such as atopic dermatitis, psoriasis and melanoma is highlighted.
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Increased FADS2-derived n-6 PUFAs and reduced n-3 PUFAs in plasma of atopic dermatitis patients.
Johanna Mihály,Tamás Marosvölgyi,Andrea Szegedi,Krisztina Köröskényi,Renata Lucas,Dániel Törőcsik,Ada L. Garcia,Tamás Decsi,Ralph Rühl,Ralph Rühl +9 more
TL;DR: It is concluded that the increased expression of FADS2 in PBMCs, as a representative tissue accessible from human blood of AD patients, might be responsible for higher levels of F ADS2-derived n-6 PUFAs and lower n-3 PUFA levels in patients.
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