Anders Bröijersén
Karolinska University Hospital
24 Papers
427 Citations
Anders Bröijersén is an academic researcher from Karolinska University Hospital. The author has contributed to research in topics: Platelet activation & Platelet. The author has an hindex of 16, co-authored 23 publications. Previous affiliations of Anders Bröijersén include Karolinska Institutet.
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Papers
Atorvastatin reduces thrombin generation and expression of tissue factor, P-selectin and GPIIIa on platelet-derived microparticles in patients with peripheral arterial occlusive disease
Fariborz Mobarrez,Shu He,Anders Bröijersén,Björn Wiklund,Aleksandra Antovic,Jovan P. Antovic,Nils Egberg,Gun Jörneskog,Håkan Wallén +8 more
TL;DR: Atorvastatin reduces thrombin generation and expression of TF, GPIIIa and P-selectin on PMPs in patients with peripheral vascular disease and in vitro experiments showed that when TF on microparticles (MPs) was blocked by antibodies, the initiation of throm bin generation was slightly but significantly delayed.
102
Influence of different anticoagulants on platelet aggregation in whole blood; a comparison between citrate, low molecular mass heparin and hirudin.
TL;DR: The selective thrombin inhibitor hirudin is the most suitable anticoagulant for studies of platelet aggregation in vitro in whole blood, reflecting the importance of extracellular calcium for platelet function.
91
Alimentary lipemia enhances the membrane expression of platelet P-selectin without affecting other markers of platelet activation
TL;DR: The present finding of enhanced platelet P-selectin expression suggests that platelets are mildly sensitized postprandially, and whether this is of importance for thrombus formation and atherosclerosis needs to be studied further.
69
On the accurate determination of serotonin in human plasma.
TL;DR: Using these procedures plasma 5-HT levels in healthy volunteers were found to be 0.77 +/- 0.38 (mean +/- S.D.; range 0.27-1.49) nmol/l, which is 4-100-fold lower than previously reported values.
62
Nitric oxide mediates protective effect of endothelin receptor antagonism during myocardial ischemia and reperfusion
TL;DR: The dual ETA/ETB receptor antagonist bosentan preserved endothelial and cardiac contractile function during ischemia and reperfusion via a mechanism dependent on endothelial NO production.
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