ackground The inverse relation between alcohol intake and clinical coronary artery disease (CAD) is well estabished, although the mechanisms remain speculative. We studied the relation between alcohol intake and subclinical CAD o assess the possible role of alcohol in atherogenesis. ethods We conducted a prospective study of 731 consecutive, consenting, active-duty US Army personnel (39 to 5 years of age) without known CAD who were undergoing a routine physical examination. Each participant was sureyed with the validated Block dietary questionnaire, which included detailed information on alcohol intake as wine, eer, or liquor. Subclinical CAD was determined by means of electron beam computed tomography to quantify coronary rtery calcification (CAC). esults The mean age was 42 ( 2); 83% were male, 71% were white, and 82% were college graduates. The prevlence of CAC was 18.6% (mean CAC score 12 69). Twenty-two percent drank alcohol daily, with an average of .4 drinks per day. Systolic blood pressure was correlated with number of drinks per day (r 0.10, P .025). Among rinkers, HDL was weakly correlated with daily alcohol consumption (r 0.10, P .025). There was no relation beween the CAC score and the alcohol intake as measured by drinks per day (OR, 1.02; 95% CI, 0.64 to 1.63; 1.13, .59 to 2.15; 1.26, 0.69 to 2.59, for less than 1, 1 to 2, and more than 2 drinks per day, respectively). Stratified analyes based on type of alcohol and multivariate analyses indicated no independent relation between any type or quantity of lcohol intake and the presence or extent of coronary calcification. onclusions Alcohol intake does not appear to be inversely related to subclinical CAC, implying that previous obervations of a protective effect of alcohol on clinical CAD may involve factors related to plaque stability rather than therogenesis. (Am Heart J 2004;148:803–9.)

lcohol intake is not associated with subclinical oronary atherosclerosis

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lcohol intake is not associated with subclinical oronary atherosclerosis