Alison Brown
Harvard University
9 Papers
Alison Brown is an academic researcher from Harvard University. The author has contributed to research in topics: Single-nucleotide polymorphism & Haplotype. The author has an hindex of 9, co-authored 9 publications. Previous affiliations of Alison Brown include Washington University in St. Louis & Brigham and Women's Hospital.
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Papers
Utilization of a whole genome SNP panel for efficient genetic mapping in the mouse
Jennifer L. Moran,Andrew D. Bolton,Pamela V. Tran,Alison Brown,Noelle D. Dwyer,Danielle K. Manning,Bryan C. Bjork,Cheng Li,Kate Montgomery,Sandra M. Siepka,Martha Hotz Vitaterna,Joseph S. Takahashi,Tim Wiltshire,David J. Kwiatkowski,Raju Kucherlapati,David R. Beier +15 more
TL;DR: A fixed single nucleotide polymorphism panel of 394 SNPs was developed as an alternative to analyses using simple sequence length polymorphism (SSLP) marker mapping to facilitate genetic mapping and identified DNA sequence changes in two ethylnitrosourea-induced mutants.
The IL12B Gene Is Associated with Asthma
Adrienne G. Randolph,Adrienne G. Randolph,Adrienne G. Randolph,Christoph Lange,Edwin K. Silverman,Edwin K. Silverman,Ross Lazarus,Ross Lazarus,Eric S. Silverman,Eric S. Silverman,Benjamin A. Raby,Benjamin A. Raby,Alison Brown,Al Ozonoff,Brent Richter,Scott T. Weiss,Scott T. Weiss +16 more
TL;DR: A strong association is found between the IL 12B_4237 and IL12B_6402 polymorphisms and an asthma-severity phenotype in whites, which is found in the independent population of white adult asthmatics and may be an important asthma gene.
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Sex differences in cell migration in the preoptic area/anterior hypothalamus of mice.
TL;DR: The significant dorsal-ventral migration of cells in the POA/AH provides additional support for the notion that the region integrates developmental information from both telencephalic and diencephalic compartments.
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The preoptic area/anterior hypothalamus of different strains of mice: sex differences and development.
TL;DR: Sex differences in neural morphology which develop within the POA/AH depend upon multiple factors, particularly including genetic background.
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Genomewide linkage analysis of quantitative spirometric phenotypes in severe early-onset chronic obstructive pulmonary disease.
Edwin K. Silverman,Edwin K. Silverman,Lyle J. Palmer,Jonathan D. Mosley,Matthew J. Barth,Jody M. Senter,Alison Brown,Jeffrey M. Drazen,Jeffrey M. Drazen,David J. Kwiatkowski,Harold A. Chapman,Edward J. Campbell,Michael A. Province,D. C. Rao,John J. Reilly,John J. Reilly,Leo C. Ginns,Frank E. Speizer,Scott T. Weiss +18 more
TL;DR: Observations provide both significant evidence for an early-onset COPD-susceptibility locus on chromosome 2 and suggestive evidence for linkage of spirometry-related phenotypes to several other genomic regions.