Alfonso Díaz
Benemérita Universidad Autónoma de Puebla
64 Papers
172 Citations
Alfonso Díaz is an academic researcher from Benemérita Universidad Autónoma de Puebla. The author has contributed to research in topics: Hippocampus & Dendritic spine. The author has an hindex of 18, co-authored 64 publications. Previous affiliations of Alfonso Díaz include Central Queensland University & National Autonomous University of Mexico.
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Papers
Vanadium in Biological Action: Chemical, Pharmacological Aspects, and Metabolic Implications in Diabetes Mellitus
Samuel Treviño,Alfonso Díaz,Eduardo Sánchez-Lara,Brenda L. Sanchez-Gaytan,Jose Manuel Perez-Aguilar,Enrique González-Vergara +5 more
TL;DR: This review examines the development of vanadium-containing compounds in biological systems regarding the role of the physiological environment, dosage, intracellular interactions, metabolic transformations, modulation of signaling pathways, toxicology, and transport and tissue distribution as well as therapeutic implications and sheds light on the prevailing gaps between primary scientific data and information from animal models and human studies.
Alzheimer's disease and metabolic syndrome: A link from oxidative stress and inflammation to neurodegeneration.
Eduardo Rojas-Gutierrez,Guadalupe Muñoz-Arenas,Samuel Treviño,Blanca Espinosa,Raúl Chávez,Karla Rojas,Gonzalo Flores,Alfonso Díaz,Jorge Guevara +8 more
TL;DR: Therapeutic efforts to achieve attenuation of oxidative stress could be beneficial in AD treatment, attenuating Aβ‐induced neurotoxicity and improve neurological outcomes in AD.
160
Reactive oxygen species: Role in carcinogenesis, cancer cell signaling and tumor progression.
Fabiola Lilí Sarmiento-Salinas,Andrea Perez-Gonzalez,Adilene Acosta-Casique,Adrián Ix-Ballote,Alfonso Díaz,Samuel Treviño,Nora Hilda Rosas-Murrieta,Lourdes Millan-PerezPeña,Paola Maycotte +8 more
TL;DR: In this paper, a review of the role of reactive oxygen species (ROS) in carcinogenesis and cancer progression is presented, with a special focus on their role in cancer cell signaling and their relationship to the most prevalent cancer risk factors.
126
Neuroprotective effect of alpha-asarone on spatial memory and nitric oxide levels in rats injected with amyloid-β(25–35)
Ilhuicamina Daniel Limón,Liliana Mendieta,Alfonso Díaz,Germán Chamorro,Blanca Espinosa,Edgar Zenteno,Jorge Guevara +6 more
TL;DR: The results suggest that alpha-asarone may protect neurons against Abeta((25-35))-caused neurotoxicity by inhibiting the effects of NO overproduction in the hippocampus and temporal cortex.
96
Aβ25-35 injection into the temporal cortex induces chronic inflammation that contributes to neurodegeneration and spatial memory impairment in rats
TL;DR: It is suggested that the chronic inflammatory reaction could contribute to the progression of Aβ25-35 toxicity and cause cognitive impairment.
75