Ahmed Mahmoud
Ghent University Hospital
31 Papers
176 Citations
Ahmed Mahmoud is an academic researcher from Ghent University Hospital. The author has contributed to research in topics: Male infertility & Sperm. The author has an hindex of 16, co-authored 31 publications.
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Papers
Combined conventional/antioxidant “Astaxanthin” treatment for male infertility: a double blind, randomized trial
TL;DR: Although the present study suggests a positive effect of Astaxanthin on sperm parameters and fertility, the results need to be confirmed in a larger trial before recommending Astxanthin for the complementary treatment of infertile men.
Chronic prostatitis and male accessory gland infection – is there an impact on male infertility (diagnosis and therapy)?
TL;DR: By means of a review of the literature and own study material the multifactorial aetiology of male infertility is discussed, extrapolate this hypothesis to male accessory gland infection (MAGI) and relate it to chronic prostatitis and its treatment.
The clinical and biologic significance of serum inhibins in subfertile men
TL;DR: Simultaneous measurement of both FSH and inhibin B identified more cases with oligozoospermia than either hormone alone and the indirect mixed antiglobin reaction (MAR) test result appears accurate in identifying subfertility.
Is inhibin B involved in the toxic effect of lead on male reproduction
TL;DR: The results of the present study suggest that the exposure of the cells of Sertoli to excessive amounts of lead results in inappropriate inhibin B overproduction that may be involved in the impairment of spermatogenesis.
Early decline of androgen levels in healthy adult men : an effect of aging per se? A prospective cohort study
Thiberiu Banica,Charlotte Verroken,Tim Reyns,Ahmed Mahmoud,Guy T'Sjoen,Tom Fiers,Jean-Marc Kaufman,Bruno Lapauw +7 more
TL;DR: In this paper, the authors investigated longitudinal changes in serum androgen levels in healthy men in relation to body composition, lifestyle factors, and intercurrent disease, and concluded that aging per se leads to an altered sex steroid status.