Ahmed Mahmoud
Ghent University Hospital
31 Papers
176 Citations
Ahmed Mahmoud is an academic researcher from Ghent University Hospital. The author has contributed to research in topics: Male infertility & Sperm. The author has an hindex of 16, co-authored 31 publications.
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Papers
Endogenous testosterone and cardiovascular disease in healthy men: a meta-analysis
TL;DR: A weak independent association was found with an estimated summary RR of 0.89 for a change of one standard deviation in total testosterone level for middle-aged men, and in elderly men, testosterone may weakly protect against CVD.
304
Testicular Volume in Relation to Hormonal Indices of Gonadal Function in Community-Dwelling Elderly Men
TL;DR: The moderately decreased BTV observed in elderly men, strongly associated with a decrease of the inhibin B/FSH ratio, is consistent with a reduced Sertoli cell mass, compensated by increased FSH stimulation resulting in only limited decrease of SERToli cell function.
159
Aging and the Male Reproductive System.
TL;DR: The benefits and risks of T therapy in older men are discussed in this paper, where the authors present an overview of current knowledge on fertility and reproductive hormone changes in aging men, the factors driving and modulating these changes, their clinical consequences, and the benefits and risk of testosterone (T) therapy.
The role of food supplements in the treatment of the infertile man
Frank Comhaire,Ahmed Mahmoud +1 more
TL;DR: Sperm quality and function improved with the intake of complementary food supplementation using a combination of zinc and folic acid, or the antioxidant astaxanthin (Astacarox), or an energy-providing combination containing (actyl)-carnitine (Proxeed).
88
The decline of serum testosterone levels in community-dwelling men over 70 years of age: descriptive data and predictors of longitudinal changes
Bruno Lapauw,Stefan Goemaere,Hans-Georg Zmierczak,I Van Pottelbergh,Ahmed Mahmoud,Youri Taes,Dirk De Bacquer,Stijn Vansteelandt,Jean Kaufman +8 more
TL;DR: The identification of FSH levels as a predictor of this decline appears to reflect the testicular mechanisms of aging-related changes in testosterone production, whereas associations with E(2) and ER alpha polymorphisms are suggestive of estrogen-related processes, possibly related to changes in the neuroendocrine regulation of testosterone production.