Adriana Albini
University of Milano-Bicocca
407 Papers
5.3K Citations
Adriana Albini is an academic researcher from University of Milano-Bicocca. The author has contributed to research in topics: Angiogenesis & Matrigel. The author has an hindex of 85, co-authored 391 publications. Previous affiliations of Adriana Albini include National Institutes of Health & University of Insubria.
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Papers
•Journal Article
A Rapid in Vitro Assay for Quantitating the Invasive Potential of Tumor Cells
Adriana Albini,Yukihide Iwamoto,Hynda K. Kleinman,George R. Martin,Stuart A. Aaronson,Kozlowski Jm,R. N. McEwan +6 more
TL;DR: The results suggest that this in vitro test allows the rapid and quantitative assessment of invasiveness and a means to screen for drugs which alter the invasive phenotype of tumor cells.
1.9K
The tumour microenvironment as a target for chemoprevention
Adriana Albini,Michael B. Sporn +1 more
TL;DR: New data indicate that primary dysfunction in the tumour microenvironment, in addition to epithelial dysfunction, can be crucial for carcinogenesis, and makes a compelling case for targeting the microenvironment for cancer chemoprevention.
854
Cardiotoxicity of Anticancer Drugs: The Need for Cardio-Oncology and Cardio-Oncological Prevention
Adriana Albini,Giuseppina Pennesi,Francesco Donatelli,Rosaria Cammarota,Silvio De Flora,Douglas M. Noonan +5 more
TL;DR: The potential cardiovascular toxicities for a range of cancer chemotherapeutic and chemopreventive agents are summarized and the importance of evaluating cardiovascular risk when patients enter into trials is emphasized and the need to develop guidelines that include collateral effects on the cardiovascular system is emphasized.
Bone marrow neovascularization, plasma cell angiogenic potential, and matrix metalloproteinase-2 secretion parallel progression of human multiple myeloma.
Angelo Vacca,Domenico Ribatti,Marco Presta,M Minischetti,Monica Iurlaro,Roberto Ria,Adriana Albini,Federico Bussolino,Franco Dammacco +8 more
TL;DR: Findings indicate that the progression of plasma cell tumors is accompanied by an increase of bone marrow neovascularization, which is dependent, at least in part, by FGF-2 and MMP-2 production.
644
The angiogenesis induced by HIV-1 tat protein is mediated by the Flk-1/KDR receptor on vascular endothelial cells.
Adriana Albini,Raffaella Soldi,Daniela Giunciuglio,Enrico Giraudo,Roberto Benelli,Luca Primo,Douglas M. Noonan,Mariolina Salio,Giovanni Camussi,Wolfang Rockl,Federico Bussolino +10 more
TL;DR: It is demonstrated that HIV–1 Tat specifically binds and activates the Flk–1/kinase insert domain receptor (Flk-1/KDR), a VEGF–A tyrosine kinase receptor, and that Tat–induced angiogenesis is blocked by agents blocking the FlK–1-KDR receptor.
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