A. Zaman
University of Pennsylvania
6 Papers
70 Citations
A. Zaman is an academic researcher from University of Pennsylvania. The author has contributed to research in topics: Phosphocreatine & Cytochrome c oxidase. The author has an hindex of 5, co-authored 6 publications.
Chat about Author
Papers
Microtiter plate assay for the measurement of glutathione and glutathione disulfide in large numbers of biological samples.
TL;DR: By combining the least complicated and expedient methods of sample handling with the sensitivity and specificity of the GSH assay by enzymatic recycling and the small volumes and software capabilities of microtiter plate technology, this work devised a rapid, sensitive, and easy assay for GSH and GSSG in biological samples.
954
The detection of cytochrome oxidase heme iron and copper absorption in the blood-perfused and blood-free brain in normoxia and hypoxia.
TL;DR: Parallel absorbance changes in the heme and copper components of cytochrome aa3 were obtained in rat and cat brains during the normoxic-hypoxic transition, indicating that the optical method responded primarily to the saturation of the venous ends of the capillary beds.
46
Seizure susceptibility during recovery from hypercapnia in neonatal dogs
Hiroshi Yoshioka,Hiroshi Yoshioka,Shoko Nioka,H. Miyake,A. Zaman,Tadashi Sawada,Britton Chance +6 more
TL;DR: In this article, the authors investigated seizure susceptibility during recovery from hypercapnia in seven anesthetized neonatal dogs; the animals were allowed to recover for 45 min; during this recovery phase, electrocorticography was performed.
12
31P magnetic resonance spectroscopy study of cerebral metabolism in developing dog brain and its relationship to neuronal function
TL;DR: Developmental increases in high energy phosphates and the maintenance of PCr/Pi indicate that the increased energy demands of the developing animal are met by increased mitochondrial function (ATP turnover).
8
Consequences of reduced cerebral blood flow in brain development. II. Retardation of neurological outcome and phosphorus metabolism.
TL;DR: The simultaneous occurrence of several biochemical and functional abnormalities in development following early ischemic insult suggests a causal relationship between membrane and mitochondrial development and neurological function.
6