A. Verna
University of Bordeaux
15 Papers
195 Citations
A. Verna is an academic researcher from University of Bordeaux. The author has contributed to research in topics: Carotid body & Free nerve ending. The author has an hindex of 11, co-authored 15 publications.
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Papers
Modifications in number and morphology of dendritic spines resulting from chronic ethanol consumption and withdrawal: A Golgi study in the mouse anterior and posterior hippocampus
TL;DR: It is demonstrated that chronic alcohol consumption leads to morphological alterations and loss of dendritic spines in the hippocampus, however, both dendrite spine number and morphology progressively return to normal values after 2 months of withdrawal.
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Build-up and release from proactive interference during chronic ethanol consumption in mice: A behavioral and neuroanatomical study
TL;DR: A neuroanatomical study was conducted to quantify cell losses resulting from 8, 24 or 48 weeks of ethanol treatment in the mammillary bodies (MM) or the hippocampus (HPC), which emphasises the importance of MM lesion in memory deficits resulting from long-term alcohol consumption.
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Chronic ethanol consumption induces neuronal loss in mammillary bodies of the mouse: A quantitative analysis
TL;DR: Quantitative analysis, using histological sections, showed that chronic ethanol consumption in the mouse produced neuronal loss in the medial mammillary bodies, in agreement with patterns of neuroanatomical damage observed in human alcoholics.
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Neuroanatomical effects of chronic ethanol consumption on dorsomedial and anterior thalamic nuclei and on substantia innominata in mice
TL;DR: Quantitative analysis, using histological sections, showed that chronic ethanol consumption for 7 months produced a weak but significant cellular loss in the dorsomedial thalamic nucleus, anterior thalamus and substantia innominata in the mouse.
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Effects of metabolic inhibitors and hypoxia on the ATP, ADP and AMP content of the rabbit carotid body in vitro: the metabolic hypothesis in question.
TL;DR: The effects of metabolic inhibitors (cyanide, antimycin) and hypoxia on the nucleotide content of the carotid body were investigated in vitro and the significance of these results is discussed with regard to the metabolic hypothesis.
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