A. Sensi
University of Ferrara
50 Papers
535 Citations
A. Sensi is an academic researcher from University of Ferrara. The author has contributed to research in topics: Gene & Antigen. The author has an hindex of 17, co-authored 49 publications.
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Papers
Mole maker phenotype: possible narrowing of the candidate region.
A. Sensi,Francesca Gualandi,Mc Pittalis,Olga Calabrese,Francesco Falciano,Iva Maestri,Luciano Bovicelli,Elisa Calzolari +7 more
TL;DR: Molecular data is provided on an additional family confirming that recurrent familial hydatidiform moles are diploid, biparental and arise from independent conceptions.
Clinical and genetic diagnosis of warts, hypogammaglobulinemia, infections, and myelokathexis syndrome in 10 patients.
Laura Tassone,Lucia Dora Notarangelo,Vanessa Bonomi,Gianfranco Savoldi,A. Sensi,Annarosa Soresina,C. I. Edvard Smith,Fulvio Porta,Alessandro Plebani,Luigi D. Notarangelo,Raffaele Badolato +10 more
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Clinical and radiological evaluation in children with microtia
TL;DR: A high correlation between the degree of microtia and the frequency of external and middle ear dysplasias was found, in accordance with larger studies of the literature.
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Chromosomal aberrations induced by BK virus T antigen in human fibroblasts.
Cecilia Trabanelli,Alfredo Corallini,R. Gruppioni,A. Sensi,A. Bonfatti,Diana Campioni,Michela Merlin,Nilla Calza,Laura Possati,Giuseppe Barbanti-Brodano +9 more
TL;DR: In this paper, the authors found that human fibroblasts, transfected with a recombinant DNA containing the neo gene and BK Virus (BKV) early region, which expresses BKV large T antigen (TAg), show cytogenetic alterations characterized by dicentric chromosomes and other structural aberrations such as deletions, duplications, translocations, and ring chromosomes.
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Induction of senescence and control of tumorigenicity in BK virus transformed mouse cells by human chromosome 6
Francesca Gualandi,Cristina Morelli,Jorge V. Pavan,Paola Rimessi,A. Sensi,A. Bonfatti,R. Gruppioni,Laura Possati,Eric J. Stanbridge,Giuseppe Barbanti-Brodano +9 more
TL;DR: The monochromosomic hybrids bearing a deleted chromosome 6 showed a reverted phenotype in vitro and a significantly longer latency period before they were tumorigenic in nude mice, indicating the presence of a tumor suppressor gene in the residual regions of chromosome 6.
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