A. D. Bocking
University of Western Ontario
36 Papers
671 Citations
A. D. Bocking is an academic researcher from University of Western Ontario. The author has contributed to research in topics: Fetus & Gestational age. The author has an hindex of 20, co-authored 36 publications. Previous affiliations of A. D. Bocking include Lawson Health Research Institute.
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Papers
Metabolic and circulatory adaptations to chronic hypoxia in the fetus.
TL;DR: Much of this information is based on experimental data using unanesthetized fetal sheep with chronic catheterization; however, clinical outcome data and the use of investigative techniques have supported the relevance of this experimental data to the human situation.
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Circulatory responses to prolonged hypoxemia in fetal sheep.
TL;DR: It is demonstrated that the sheep fetus is able to maintain the normal protective circulatory adjustments seen with acute hypoxemia for up to 48 hours in the absence of progressive metabolic acidemia.
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Preterm discordant twins: what birth weight difference is significant?
TL;DR: In preterm twin gestations the use of a 30% birth weight difference to define twin discordance is most clinically relevant in identifying those infants at risk for adverse perinatal outcome.
104
15-hydroxyprostaglandin dehydrogenase: implications in preterm labor with and without ascending infection.
Claudia A. van Meir,Stephen G. Matthews,Stephen G. Matthews,Marc J. N. C. Keirse,M. M. Ramirez,A. D. Bocking,John R. G. Challis,John R. G. Challis +7 more
TL;DR: A reduction in PGDH in the human fetal membranes may occur in some cases of preterm labor and may contribute to an increase in net PG accumulation and drive to myometrial contractility.
Extracellular glutamate levels and neuropathology in cerebral white matter following repeated umbilical cord occlusion in the near term fetal sheep
Michelle Loeliger,Carole S. Watson,James D. Reynolds,Donald H. Penning,Richard Harding,A. D. Bocking,Sandra Rees +6 more
TL;DR: In the unanesthetised fetus in utero, glutamatergic processes are associated with umbilical cord occlusion-induced brain damage in the cerebral white matter.
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